← back to search

Cynthia J. Tifft

University of Maryland, College Park · US
🔎 Find collaborators in Physiology · Genetics →
Search 5.9M scientists by topic, h-index, country & funding — free.
Area of research
Physiology · Genetics
Research interest
Research interests include Medicine, Biology, Genetics, Genetic enhancement, Disease, and Proband.
h-index
citations
3,273
works
25
NIH funding
primary concept
email

Recent publications

An optimized variant prioritization process for rare disease diagnostics: recommendations for Exomiser and Genomiser
Genome Medicine 2025cited by 7position: middledoi
AAV9 Gene Therapy in GM1 Gangliosidosis Type II: A Phase 1/2 Trial
medRxiv 2025cited by 2position: lastdoi
De novo variants in the RNU4-2 snRNA cause a frequent neurodevelopmental syndrome
Nature 2024cited by 112position: middledoi
A pentasaccharide for monitoring pharmacodynamic response to gene therapy in GM1 gangliosidosis
EBioMedicine 2023cited by 18position: middledoi
Mechanism of KMT5B haploinsufficiency in neurodevelopment in humans and mice
Science Advances 2023cited by 17position: middledoi
AAV gene therapy for Tay-Sachs disease
Nature Medicine 2022cited by 134position: middledoi
Gene-based therapeutics for rare genetic neurodevelopmental psychiatric disorders
Molecular Therapy 2022cited by 24position: middledoi
Clinical and molecular features of 66 patients with musculocontractural Ehlers−Danlos syndrome caused by pathogenic variants in <i>CHST14</i> (mcEDS-<i>CHST14</i>)
Journal of Medical Genetics 2021cited by 41position: middledoi
Distinct interferon signatures and cytokine patterns define additional systemic autoinflammatory diseases
Journal of Clinical Investigation 2019cited by 233position: middledoi
Lysosomal storage diseases
Nature Reviews Disease Primers 2018cited by 983position: lastdoi
Effect of Genetic Diagnosis on Patients with Previously Undiagnosed Disease
New England Journal of Medicine 2018cited by 369position: middledoi
A Recurrent De Novo Heterozygous COG4 Substitution Leads to Saul-Wilson Syndrome, Disrupted Vesicular Trafficking, and Altered Proteoglycan Glycosylation
The American Journal of Human Genetics 2018cited by 73position: middledoi
De Novo Missense Variants in TRAF7 Cause Developmental Delay, Congenital Anomalies, and Dysmorphic Features
The American Journal of Human Genetics 2018cited by 51position: middledoi
Novel mutations in CLN6 cause late-infantile neuronal ceroid lipofuscinosis without visual impairment in two unrelated patients
Molecular Genetics and Metabolism 2018cited by 17position: middledoi
Functional performance in late-onset GM2 gangliosidosis (Tay-Sachs and Sandhoff diseases), longitudinal data over 3 consecutive years
Molecular Genetics and Metabolism 2018cited by 1position: middledoi
The Undiagnosed Diseases Network: Accelerating Discovery about Health and Disease
The American Journal of Human Genetics 2017cited by 187position: middledoi
Novel Biomarkers of Human GM1 Gangliosidosis Reflect the Clinical Efficacy of Gene Therapy in a Feline Model
Molecular Therapy 2017cited by 57position: middledoi
Defining Disease, Diagnosis, and Translational Medicine within a Homeostatic Perturbation Paradigm: The National Institutes of Health Undiagnosed Diseases Program Experience
Frontiers in Medicine 2017cited by 28position: middledoi
A Syndromic Neurodevelopmental Disorder Caused by De Novo Variants in EBF3
The American Journal of Human Genetics 2016cited by 121position: middledoi
De novo missense variants in <i>HECW2</i> are associated with neurodevelopmental delay and hypotonia
Journal of Medical Genetics 2016cited by 63position: middledoi
Recurrent Mutations in the Basic Domain of TWIST2 Cause Ablepharon Macrostomia and Barber-Say Syndromes
The American Journal of Human Genetics 2015cited by 76position: middledoi
<i>GRIN2A</i> mutation and early‐onset epileptic encephalopathy: personalized therapy with memantine
Annals of Clinical and Translational Neurology 2014cited by 295position: middledoi
Three rare diseases in one Sib pair: RAI1, PCK1, GRIN2B mutations associated with Smith–Magenis Syndrome, cytosolic PEPCK deficiency and NMDA receptor glutamate insensitivity
Molecular Genetics and Metabolism 2014cited by 65position: middledoi
Multilineage somatic activating mutations in HRAS and NRAS cause mosaic cutaneous and skeletal lesions, elevated FGF23 and hypophosphatemia
Human Molecular Genetics 2013cited by 135position: middledoi
Detecting false-positive signals in exome sequencing
Human Mutation 2012cited by 164position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

William A. Gahl · National Institutes of Health5 papers (2012–2018)Heather Gray Edwards · Auburn University4 papers (2017–2023)David R. Adams · National Institutes of Health4 papers (2012–2018)Gretchen Golas · National Human Genome Research Institute4 papers (2013–2017)Camilo Toro · National Institutes of Health4 papers (2012–2018)Cornelius F. Boerkoel · Hospital for Sick Children3 papers (2012–2014)Thomas C. Markello · National Human Genome Research Institute3 papers (2012–2014)Beverly L. Davidson · Children's Hospital of Philadelphia2 papers (2018–2022)Carsten G. Bönnemann · National Institutes of Health2 papers (2016–2022)Murat Sincan · National Human Genome Research Institute2 papers (2012–2014)Karin Fuentes Fajardo · National Institutes of Health2 papers (2014–2014)Lynne A. Wolfe · National Human Genome Research Institute2 papers (2014–2016)Hongjie Yuan · Emory University2 papers (2014–2014)Allison M. Bradbury · Nationwide Children's Hospital2 papers (2017–2022)Douglas R. Martin · Auburn University2 papers (2017–2023)Jean M. Johnston · National Human Genome Research Institute2 papers (2017–2018)Conisha Holloman · University of Rochester2 papers (2014–2014)Stephen F. Traynelis · Emory University2 papers (2014–2014)Mariska Davids · National Human Genome Research Institute1 papers (2018–2018)Tyler Mark Pierson · Cedars-Sinai Medical Center1 papers (2014–2014)
Looking for a research collaborator?
Search millions of scientists by field, institution, impact, and funding status — see their work, find their email, and reach out directly.
Find collaborators in Physiology · Genetics →