Area of research
Physiology · Epidemiology
Research interest
Research interests include Adipose Tissue and Metabolism, Adipokines, Inflammation, and Metabolic Diseases, Birth, Development, and Health, and Gestational Diabetes Research and Management.
Maternal Obesity Programs Glucose Intolerance in Pregnant Female Offspring.
Maternal GLP-1 receptor activation inhibits fetal growth
Maternal GLP-1 receptor activation inhibits fetal growth.
The Essential Role of Pancreatic α-Cells in Maternal Metabolic Adaptation to Pregnancy.
Adiponectin Promotes Maternal β-Cell Expansion Through Placental Lactogen Expression.
Adiponectin Promotes Maternal β-Cell Expansion Through Placental Lactogen Expression
Obesity Reduces Maternal Blood Triglyceride Concentrations by Reducing Angiopoietin-Like Protein 4 Expression in Mice.
Prolonged Prepregnant Maternal High-Fat Feeding Reduces Fetal and Neonatal Blood Glucose Concentrations by Enhancing Fetal β-Cell Development in C57BL/6 Mice.
The platelet-derived growth factor receptor alpha promoter-directed expression of cre recombinase in mouse placenta.
High-fat feeding reprograms maternal energy metabolism and induces long-term postpartum obesity in mice.
Adiponectin Deficiency Impairs Maternal Metabolic Adaptation to Pregnancy in Mice
Maternal High-Fat Feeding Increases Placental Lipoprotein Lipase Activity by Reducing SIRT1 Expression in Mice
The PARsylation activity of tankyrase in adipose tissue modulates systemic glucose metabolism in mice
Muscle wasting and adipose tissue browning in infantile nephropathic cystinosis
Nutritional Energy Stimulates NAD+ Production to Promote Tankyrase-Mediated PARsylation in Insulinoma Cells
Cytoplasmic Tyrosine Phosphatase Shp2 Coordinates Hepatic Regulation of Bile Acid and FGF15/19 Signaling to Repress Bile Acid Synthesis
Adiponectin reduces thermogenesis by inhibiting brown adipose tissue activation in mice
Adiponectin and energy homeostasis
Adiponectin Increases Skeletal Muscle Mitochondrial Biogenesis by Suppressing Mitogen-Activated Protein Kinase Phosphatase-1