Area of research
Oncology · Molecular Biology
Research interest
Research interests include Drug Transport and Resistance Mechanisms, Ion Transport and Channel Regulation, Helicobacter pylori-related gastroenterology studies, and Digestive system and related health.
A Direct Link Implicating Loss of SLC26A6 to Gut Microbial Dysbiosis, Compromised Barrier Integrity, and Inflammation
Serum metabolomic analysis in cirrhotic alcohol-associated liver disease patients identified differentially altered microbial metabolites and novel potential biomarkers for disease severity
Mucosal Metabolomic Signatures in Chronic Colitis: Novel Insights into the Pathophysiology of Inflammatory Bowel Disease
Potential Dietary and Therapeutic Strategies Involving Indole-3-Carbinole in Preclinical Models of Intestinal Inflammation
Bile acids as inflammatory mediators and modulators of intestinal permeability
A Novel Role of SLC26A3 in the Maintenance of Intestinal Epithelial Barrier Integrity
Disturbances in Cholesterol Homeostasis and Non-alcoholic Fatty Liver Diseases
Serum Serotonin Differentiates Between Disease Activity States in Crohn’s Patients
Intestinal Absorption of Bile Acids in Health and Disease
<i>Cryptosporidium parvum</i> infection induces autophagy in intestinal epithelial cells
S-acylation modulates the function of the apical sodium-dependent bile acid transporter in human cells
Bile acid receptors and gastrointestinal functions
Serotonin Transporter Deficiency is Associated with Dysbiosis and Changes in Metabolic Function of the Mouse Intestinal Microbiome
Serotonin Modulates AhR Activation by Interfering with CYP1A1-Mediated Clearance of AhR Ligands
Recent advances in understanding and managing malabsorption: focus on microvillus inclusion disease
All-trans Retinoic Acid Counteracts Diarrhea and Inhibition of Downregulated in Adenoma Expression in Gut Inflammation
miR-125a-5p: a novel regulator of SLC26A6 expression in intestinal epithelial cells
Phospholipid Remodeling and Cholesterol Availability Regulate Intestinal Stemness and Tumorigenesis
Pathophysiology of IBD associated diarrhea
Serotonin is an endogenous regulator of intestinal CYP1A1 via AhR
<i>Cryptosporidium parvum</i>disrupts intestinal epithelial barrier function via altering expression of key tight junction and adherens junction proteins
<i>Clostridium difficile</i> toxins A and B decrease intestinal SLC26A3 protein expression
Activation of Nuclear Factor−κB by Tumor Necrosis Factor in Intestinal Epithelial Cells and Mouse Intestinal Epithelia Reduces Expression of the Chloride Transporter SLC26A3
Expression and localization of VPAC1, the major receptor of vasoactive intestinal peptide along the length of the intestine
Role of SHP2 protein tyrosine phosphatase in SERT inhibition by enteropathogenic<i>E. coli</i>(EPEC)
CDX2 upregulates SLC26A3 gene expression in intestinal epithelial cells
A novel anti-inflammatory role of GPR120 in intestinal epithelial cells
<i>Lactobacillus acidophilus</i> counteracts inhibition of NHE3 and DRA expression and alleviates diarrheal phenotype in mice infected with <i>Citrobacter rodentium</i>
<i>Lactobacillus acidophilus</i>stimulates intestinal P-glycoprotein expression via a c-Fos/c-Jun-dependent mechanism in intestinal epithelial cells
Mechanisms Underlying Dysregulation of Electrolyte Absorption in Inflammatory Bowel Disease–Associated Diarrhea