Area of research
Reproductive Medicine · Immunology
Research interest
Research interests include Cell biology, Cancer research, Endometriosis, Chemistry, Stromal cell, and MAPK/ERK pathway.
Elevated Histone Lactylation Mediates Ferroptosis Resistance in Endometriosis Through the METTL3‐Regulated HIF1A/HMOX1 Signaling Pathway
Correction: Macrophages originated IL-33/ST2 inhibits ferroptosis in endometriosis via the ATF3/SLC7A11 axis
IGF2BP3 promotes glutamine metabolism of endometriosis by interacting with UCA1 to enhances the mRNA stability of GLS1
Macrophages originated IL-33/ST2 inhibits ferroptosis in endometriosis via the ATF3/SLC7A11 axis
Ectopic endometriotic stromal cells‐derived lactate induces <scp>M2</scp> macrophage polarization via Mettl3/Trib1/<scp>ERK</scp>/<scp>STAT3</scp> signalling pathway in endometriosis
Silencing of lncRNA MALAT1 facilitates erastin-induced ferroptosis in endometriosis through miR-145-5p/MUC1 signaling
FTO-dependent N(6)-Methyladenosine regulates the progression of endometriosis via the ATG5/PKM2 Axis
CCL20/CCR6 axis mediates macrophages to promote proliferation and migration of ESCs by blocking autophagic flux in endometriosis