Area of research
Sensory Systems · Molecular Biology
Research interest
Research interests include Ion Channels and Receptors, Ion channel regulation and function, Neurofibromatosis and Schwannoma Cases, and Pain Mechanisms and Treatments.
Molecular mechanism of menthol-induced TRPV5 channel inhibition.
Structural basis of the inhibition of TRPV1 by analgesic sesquiterpenes.
Roles for TRPV4 in disease: A discussion of possible mechanisms.
Permeant cations modulate pore dynamics and gating of TRPV1 ion channels.
Identification and Properties of TRPV4 Mutant Channels Present in Polycystic Kidney Disease Patients.
Modes of action of lysophospholipids as endogenous activators of the TRPV4 ion channel
Modes of action of lysophospholipids as endogenous activators of the TRPV4 ion channel.
Unconventional interactions of the TRPV4 ion channel with beta-adrenergic receptor ligands.
Cation permeability and pore dynamics in TRPV1 ion channels
TRP channels: a journey towards a molecular understanding of pain.
Epithelia-Sensory Neuron Cross Talk Underlies Cholestatic Itch Induced by Lysophosphatidylcholine
TRPV4 activity regulates nuclear Ca<sup>2+</sup> and transcriptional functions of β-catenin in a renal epithelial cell model.
Discovery and characterization of H <sub>v</sub> 1-type proton channels in reef-building corals
TRPV1: Structure, Endogenous Agonists, and Mechanisms.
TRPV4: A Physio and Pathophysiologically Significant Ion Channel.
The Contribution of the Ankyrin Repeat Domain of TRPV1 as a Thermal Module.
Steroids and TRP Channels: A Close Relationship.
TRPV1 Channel: A Noxious Signal Transducer That Affects Mitochondrial Function.
KV1.2 channels inactivate through a mechanism similar to C-type inactivation.
Molecular Interplay Between the Sigma-1 Receptor, Steroids, and Ion Channels.
Cholesterol as a Key Molecule That Regulates TRPV1 Channel Function.
TRP ion channels: Proteins with conformational flexibility.
K <sub>V</sub> 1.2 channels inactivate through a mechanism similar to C-type inactivation
Connections between constitutional mismatch repair deficiency syndrome and neurofibromatosis type 1