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Lu Wen

Union Hospital · CN
Area of research
Molecular Biology · Pathology and Forensic Medicine
Research interest
Research interests include Single-cell and spatial transcriptomics, Epigenetics and DNA Methylation, Genetic factors in colorectal cancer, and Ferroptosis and cancer prognosis.
h-index
58
citations
16,606
works
380
NIH funding
primary concept
Medicine
email

Recent publications

Single-cell omics sequencing technologies: the long-read generation.
2026cited by 3position: contributordoi
A targeted DNA methylation method for detecting gastrointestinal cancer in circulating cell-free DNA
iScience 2026cited by 0position: contributordoi
Chromatin accessibility landscape of mouse early embryos revealed by single-cell NanoATAC-seq2
Science 2025cited by 25position: middledoi
Chromatin accessibility landscape of mouse early embryos revealed by single-cell NanoATAC-seq2.
2025cited by 11position: contributordoi
Systematic characterization of full-length RNA isoforms in human colorectal cancer at single-cell resolution.
2025cited by 1position: contributordoi
Drug repurposing screening and mechanism analysis based on human colorectal cancer organoids.
2024cited by 82position: contributordoi
In-depth organic mass cytometry reveals differential contents of 3-hydroxybutanoic acid at the single-cell level.
2024cited by 18position: contributordoi
Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers.
2024cited by 17position: contributordoi
scNanoSeq-CUT&Tag: a single-cell long-read CUT&Tag sequencing method for efficient chromatin modification profiling within individual cells.
2024cited by 13position: contributordoi
High-resolution single-cell transcriptomic survey of cardiomyocytes from patients with hypertrophic cardiomyopathy.
2024cited by 10position: contributordoi
Integrative analysis of transcriptome, DNA methylome, and chromatin accessibility reveals candidate therapeutic targets in hypertrophic cardiomyopathy.
2024cited by 9position: contributordoi
Simultaneous de novo calling and phasing of genetic variants at chromosome-scale using NanoStrand-seq.
2024cited by 3position: contributordoi
[Efficacy and safety of first-line treatment with anti-CD38 monoclonal antibody-based regimen for primary plasma cell leukemia].
PubMed 2024cited by 1position: middledoi
A human fetal cerebellar map of the late second trimester reveals developmental molecular characteristics and abnormality in trisomy 21.
2024cited by 1position: contributordoi
Supplementary Figure 5 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 7 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Figure 3 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Figure 9 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 9 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 6 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Figure 14 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 5 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Figure 10 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 9 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 1 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Figure 4 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Figure 11 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 2 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Figure 7 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi
Supplementary Table 7 from Single-Cell Chromatin Accessibility Analysis Reveals the Epigenetic Basis and Signature Transcription Factors for the Molecular Subtypes of Colorectal Cancers
2024cited by 0position: contributordoi

Grants

No grants ingested yet.

Frequent collaborators

Fuchou Tang · Peking University125 papers (2013–2025) · 92 papers (2020–2025) · 90 papers (2020–2026)Haoling Xie · Peking University64 papers (2022–2024)Wei Fu · Merck & Co., Inc., Rahway, NJ, USA (United States)59 papers (2018–2025)Zhenyu Liu · Peking University54 papers (2022–2025) · 51 papers (2024–2025)Kexuan Chen · Peking University51 papers (2023–2025) · 50 papers (2024–2025)Yuqiong Hu · Ministry of Education of the People's Republic of China49 papers (2024–2024)Yuxian Bai · Harbin Medical University20 papers (2018–2021)Jie Qiao · Nanjing Normal University20 papers (2013–2023)Yi Ba · Henan Cancer Hospital19 papers (2018–2021)Wenying Deng · Regeneron (United States)18 papers (2018–2021)Junsheng Wang · Qingdao University18 papers (2018–2021)Jianwei Yang · UCLA Health18 papers (2018–2021) · 18 papers (2018–2021)Yifu He · Ministry of Education18 papers (2018–2021) · 18 papers (2018–2021)Mengyao Li · Shanxi Medical University17 papers (2022–2025)