Area of research
Physiology · Cell Biology
Research interest
Research interests include Nitric Oxide and Endothelin Effects, Cellular Mechanics and Interactions, S100 Proteins and Annexins, and Cell Adhesion Molecules Research.
Studying the Pulmonary Endothelium in Health and Disease: An Official American Thoracic Society Workshop Report
Endothelial Cell Mechano-Metabolomic Coupling to Disease States in the Lung Microvasculature
Mechanosensing and Mechanoregulation of Endothelial Cell Functions
Elevated truncated oxidized phospholipids as a factor exacerbating ALI in the aging lungs
Role of truncated oxidized phospholipids in acute endothelial barrier dysfunction caused by particulate matter
Experimental Lung Injury Reduces Krüppel-like Factor 2 to Increase Endothelial Permeability via Regulation of RAPGEF3–Rac1 Signaling
Regulation of lung endothelial permeability and inflammatory responses by prostaglandin A2: role of EP4 receptor
Effects of prostaglandin lipid mediators on agonist-induced lung endothelial permeability and inflammation
Activation of Vascular Endothelial Growth Factor (VEGF) Receptor 2 Mediates Endothelial Permeability Caused by Cyclic Stretch
Dual role of vinculin in barrier-disruptive and barrier-enhancing endothelial cell responses
Synthetic Amphipathic Helical Peptides Targeting CD36 Attenuate Lipopolysaccharide-Induced Inflammation and Acute Lung Injury
Selective Role of Vinculin in Contractile Mechanisms of Endothelial Permeability
Alterations of lung microbiota in a mouse model of LPS-induced lung injury
Mechanosensitive PPAP2B Regulates Endothelial Responses to Atherorelevant Hemodynamic Forces
ACD toxin–produced actin oligomers poison formin-controlled actin polymerization
Oxidized phospholipids protect against lung injury and endothelial barrier dysfunction caused by heat-inactivated <i>Staphylococcus aureus</i>
Gap Junction Protein Connexin43 Exacerbates Lung Vascular Permeability
GRP78 is a novel receptor initiating a vascular barrier protective response to oxidized phospholipids
Prostacyclin post-treatment improves LPS-induced acute lung injury and endothelial barrier recovery via Rap1
Attenuation of Lipopolysaccharide-Induced Lung Vascular Stiffening by Lipoxin Reduces Lung Inflammation
Paxillin mediates stretch‐induced Rho signaling and endothelial permeability <i>via</i> assembly of paxillin‐p42/44MAPK‐GEF‐H1 complex
Endothelial barrier disruption and recovery is controlled by substrate stiffness
Rap-afadin axis in control of Rho signaling and endothelial barrier recovery
Interaction of p190RhoGAP with C-terminal Domain of p120-catenin Modulates Endothelial Cytoskeleton and Permeability
Fragmented oxidation products define barrier disruptive endothelial cell response to OxPAPC
Mechanical induction of group V phospholipase A<sub>2</sub>causes lung inflammation and acute lung injury
Mechanical Forces Stimulate Endothelial Microparticle Generation via Caspase‐Dependent Apoptosis‐Independent Mechanism
Role of Phospholipid Oxidation Products in Atherosclerosis
Measurement of local permeability at subcellular level in cell models of agonist- and ventilator-induced lung injury
Stimulation of Rho signaling by pathologic mechanical stretch is a “second hit” to Rho-independent lung injury induced by IL-6