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Michael Ristow

ETH Zurich · CH
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Area of research
Molecular Biology · Aging
Research interest
Research interests include Mitochondrial Function and Pathology, Genetics, Aging, and Longevity in Model Organisms, Adipose Tissue and Metabolism, and Genetic Neurodegenerative Diseases.
h-index
65
citations
25,996
works
241
NIH funding
primary concept
email

Recent publications

Enhancing Late‐Life Survival and Mobility via <i>Mitohormesis</i> by Reducing Mitochondrial Calcium Levels
Aging Cell 2025cited by 1position: middledoi
Assessment of genetic diversity among seed transfer zones for multiple grassland plant species across Germany
Basic and Applied Ecology 2024cited by 12position: middledoi
Lithium treatment extends human lifespan: findings from the UK Biobank
Aging 2023cited by 18position: lastdoi
Evolutionarily conserved transcription factors as regulators of longevity and targets for geroprotection
Physiological Reviews 2022cited by 36position: lastdoi
Ingestion of single guide RNAs induces gene overexpression and extends lifespan in Caenorhabditis elegans via CRISPR activation
Journal of Biological Chemistry 2022cited by 15position: lastdoi
Natural products in drug discovery: advances and opportunities
Nature Reviews Drug Discovery 2021cited by 4,855position: middledoi
Current research in biotechnology: Exploring the biotech forefront
Current Research in Biotechnology 2019cited by 28position: middledoi
Animal models of obesity and diabetes mellitus
Nature Reviews Endocrinology 2018cited by 832position: middledoi
Aging and drug discovery
Aging 2018cited by 35position: middledoi
Ageing with elegans: a research proposal to map healthspan pathways
Biogerontology 2016cited by 39position: middledoi
Role of sirtuins in lifespan regulation is linked to methylation of nicotinamide
Nature Chemical Biology 2013cited by 238position: lastdoi
Factors driving plant rarity in dry grasslands on different spatial scales: a functional trait approach
Biodiversity and Conservation 2013cited by 24position: lastdoi
Impaired Insulin/IGF1 Signaling Extends Life Span by Promoting Mitochondrial L-Proline Catabolism to Induce a Transient ROS Signal
Cell Metabolism 2012cited by 428position: lastdoi

Grants

No grants ingested yet.

Frequent collaborators

Kim Zarse · Humboldt-Universität zu Berlin4 papers (2012–2025) · 2 papers (2022–2022) · 2 papers (2012–2013)Collin Y. Ewald · Harvard Stem Cell Institute2 papers (2018–2022)Fabian Fischer · ETH Zurich2 papers (2022–2022) · 2 papers (2012–2013) · 2 papers (2022–2022) · 2 papers (2012–2013) · 2 papers (2012–2013) · 2 papers (2012–2013)Stefan Priebe · University of London1 papers (2013–2013) · 1 papers (2018–2018) · 1 papers (2025–2025)Garik V. Mkrtchyan · California Institute for Regenerative Medicine1 papers (2018–2018) · 1 papers (2016–2016)Markus Perola · California Southern University1 papers (2016–2016)C. Ronald Kahn · Harvard University1 papers (2012–2012) · 1 papers (2013–2013)Timo D. Müller · Technical University of Munich1 papers (2018–2018) · 1 papers (2025–2025)
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