Kevin A. Ingle

Vascular remodeling in arteriovenous fistula treated with PDE5A inhibitors

Dually Responsive Poly(N-vinylcaprolactam)-b-poly(dimethylsiloxane)-b-poly(N-vinylcaprolactam) Polymersomes for Controlled Delivery

Activation of EP4 receptor limits transition of acute to chronic heart failure in lipoxygenase deficient mice

Cardiac changes following arteriovenous fistula creation in a mouse model

Temperature-Responsive Polymersomes of Poly(3-methyl-<i>N</i>-vinylcaprolactam)-<i>block</i>-poly(<i>N</i>-vinylpyrrolidone) To Decrease Doxorubicin-Induced Cardiotoxicity

Splenic leukocytes define the resolution of inflammation in heart failure

Genetic deletion of 12/15 lipoxygenase promotes effective resolution of inflammation following myocardial infarction

Comprehensive targeted and non-targeted lipidomics analyses in failing and non-failing heart

Resolution Agonist 15-epi-Lipoxin A4 Programs Early Activation of Resolving Phase in Post-Myocardial Infarction Healing

Excess ω-6 fatty acids influx in aging drives metabolic dysregulation, electrocardiographic alterations, and low-grade chronic inflammation

Heart functional and structural compendium of cardiosplenic and cardiorenal networks in acute and chronic heart failure pathology

Interaction of 12/15-lipoxygenase with fatty acids alters the leukocyte kinetics leading to improved postmyocardial infarction healing

Aging dysregulates D- and E-series resolvins to modulate cardiosplenic and cardiorenal network following myocardial infarction

Resolvin D1 activates the inflammation resolving response at splenic and ventricular site following myocardial infarction leading to improved ventricular function

Cardiomyocyte-specific Bmal1 deletion in mice triggers diastolic dysfunction, extracellular matrix response, and impaired resolution of inflammation

Obesity superimposed on aging magnifies inflammation and delays the resolving response after myocardial infarction