Area of research
Neurology · Physiology
Research interest
Research interests include Alzheimer's disease research and treatments, Neuroinflammation and Neurodegeneration Mechanisms, Neurological Disease Mechanisms and Treatments, and Nitric Oxide and Endothelin Effects.
A cell-autonomous role for border-associated macrophages in ApoE4 neurovascular dysfunction and susceptibility to white matter injury
Meningeal interleukin-17-producing T cells mediate cognitive impairment in a mouse model of salt-sensitive hypertension
Border-associated macrophages promote cerebral amyloid angiopathy and cognitive impairment through vascular oxidative stress
Hypoxia Inducible Factor-1α binds and activates γ-secretase for Aβ production under hypoxia and cerebral hypoperfusion
Causes and consequences of baseline cerebral blood flow reductions in Alzheimer’s disease
Tau induces PSD95–neuronal NOS uncoupling and neurovascular dysfunction independent of neurodegeneration
tPA Deficiency Underlies Neurovascular Coupling Dysfunction by Amyloid-β
Neutrophil adhesion in brain capillaries reduces cortical blood flow and impairs memory function in Alzheimer’s disease mouse models
Apoε4 disrupts neurovascular regulation and undermines white matter integrity and cognitive function
Brain Perivascular Macrophages Initiate the Neurovascular Dysfunction of Alzheimer Aβ Peptides
Brain perivascular macrophages: characterization and functional roles in health and disease
Hypertension enhances A <i>β</i> -induced neurovascular dysfunction, promotes <i>β</i> -secretase activity, and leads to amyloidogenic processing of APP
Endothelial Dysfunction and Amyloid-β-Induced Neurovascular Alterations
Water Deprivation Induces Neurovascular and Cognitive Dysfunction through Vasopressin-Induced Oxidative Stress
Innate immunity receptor CD36 promotes cerebral amyloid angiopathy