Area of research
Obstetrics and Gynecology · Reproductive Medicine
Research interest
Research interests include Uterine Myomas and Treatments, Endometriosis Research and Treatment, Neuroblastoma Research and Treatments, and Renal and related cancers.
Comprehensive Review of Uterine Leiomyosarcoma: Pathogenesis, Diagnosis, Prognosis, and Targeted Therapy
Empowering Strategies for Lifestyle Interventions, Diet Modifications, and Environmental Practices for Uterine Fibroid Prevention; Unveiling the LIFE UP Awareness
Endoplasmic reticulum stress-MMRN1 positive feedback contributes to cisplatin resistance in small cell lung cancer
Update on the Role and Regulatory Mechanism of Extracellular Matrix in the Pathogenesis of Uterine Fibroids
Current and Emerging Treatment Options for Uterine Fibroids
The Functional Role and Regulatory Mechanism of FTO m6A RNA Demethylase in Human Uterine Leiomyosarcoma
Evidence-Based Approach for Secondary Prevention of Uterine Fibroids (The ESCAPE Approach)
The Regulatory Functions and the Mechanisms of Long Non-Coding RNAs in Cervical Cancer
Simvastatin inhibits stem cell proliferation in human leiomyoma via TGF‐β3 and Wnt/β‐Catenin pathways
Human Myometrial and Uterine Fibroid Stem Cell-Derived Organoids for Intervening the Pathophysiology of Uterine Fibroid
The Functional Role and Regulatory Mechanism of Bromodomain-Containing Protein 9 in Human Uterine Leiomyosarcoma
Targeting Class I Histone Deacetylases in Human Uterine Leiomyosarcoma
Epigenetic Regulation in Uterine Fibroids—The Role of Ten-Eleven Translocation Enzymes and Their Potential Therapeutic Application
Comprehensive Review of Uterine Fibroids: Developmental Origin, Pathogenesis, and Treatment
Understanding the Impact of Uterine Fibroids on Human Endometrium Function
Simvastatin modulates estrogen signaling in uterine leiomyoma via regulating receptor palmitoylation, trafficking and degradation
The role of endocrine-disrupting chemicals in uterine fibroid pathogenesis
Vitamin D3 Ameliorates DNA Damage Caused by Developmental Exposure to Endocrine Disruptors in the Uterine Myometrial Stem Cells of Eker Rats
Targeting Hedgehog Pathway and DNA Methyltransferases in Uterine Leiomyosarcoma Cells
The Role of Hedgehog Pathway in Female Cancers
Uterine fibroids in menopause and perimenopause
Activation of β-Catenin Signaling and its Crosstalk With Estrogen and Histone Deacetylases in Human Uterine Fibroids
Myometrial progesterone hyper-responsiveness associated with increased risk of human uterine fibroids
Alcohol Consumption and Risk of Uterine Fibroids
Hypovitaminosis D exacerbates the DNA damage load in human uterine fibroids, which is ameliorated by vitamin D3 treatment
Endocrine disruptor exposure during development increases incidence of uterine fibroids by altering DNA repair in myometrial stem cells†
1,25 Dihydroxyvitamin D3 Enhances the Antifibroid Effects of Ulipristal Acetate in Human Uterine Fibroids
The Emerging Spectrum of Early Life Exposure-Related Inflammation and Epigenetic Therapy
A Preliminary Study: Human Fibroid Stro-1+/CD44+ Stem Cells Isolated From Uterine Fibroids Demonstrate Decreased DNA Repair and Genomic Integrity Compared to Adjacent Myometrial Stro-1+/CD44+ Cells
Vitamin D, a promising natural compound with anti-uterine fibroid characteristics