Area of research
Biological Psychiatry · Molecular Biology
Research interest
Research interests include Neuroscience, Medicine, Cancer research, Methamphetamine, Neuroinflammation, and Biology.
Epigenetic Mechanisms Governing Nrf2 Expression and Its Role in Ferroptosis
AMPKα2 attenuates doxorubicin induced ferroptosis by promoting NCOA4 degradation in triple negative breast cancer
An update: epigenetic mechanisms underlying methamphetamine addiction
The role of macroautophagy in substance use disorders
Inhibition of GSDMD-dependent pyroptosis decreased methamphetamine self-administration in rats
Cope with copper: From copper linked mechanisms to copper-based clinical cancer therapies
m6A modified BACE1-AS contributes to liver metastasis and stemness-like properties in colorectal cancer through TUFT1 dependent activation of Wnt signaling
Antidepressant effect of bright light therapy on patients with Alzheimer’s disease and their caregivers
Targeting neuroinflammation in Alzheimer’s disease: from mechanisms to clinical applications
The Significance of NLRP Inflammasome in Neuropsychiatric Disorders
RETRACTED: The effect of the NLRP1 inflammasome on methamphetamine-induced cognitive impairment in rats
Evidence and perspectives of cell senescence in neurodegenerative diseases
A Polyaniline Nanoparticles Crosslinked Hydrogel with Excellent Photothermal Antibacterial and Mechanical Properties for Wound Dressing
Stem Cell Therapies in Alzheimer’s Disease: Applications for Disease Modeling
Oncogenic lncRNA ZNF561-AS1 is essential for colorectal cancer proliferation and survival through regulation of miR-26a-3p/miR-128-5p-SRSF6 axis
CDK5RAP2 loss-of-function causes premature cell senescence via the GSK3β/β-catenin-WIP1 pathway
The Neuroprotective and Neurodegeneration Effects of Heme Oxygenase-1 in Alzheimer’s Disease
Heme Oxygenase 1 Inhibits Adult Neural Stem Cells Proliferation and Survival via Modulation of Wnt/β-Catenin Signaling
Adipose-derived stem cells: Sources, potency, and implications for regenerative therapies
Heme Oxygenase 1 Induces Tau Oligomer Formation and Synapse Aberrations in Hippocampal Neurons
Overexpression of Heme Oxygenase 1 Impairs Cognitive Ability and Changes the Plasticity of the Synapse