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Igor Kramnik

Boston University · US
Area of research
Infectious Diseases · Epidemiology
Research interest
Research interests include Tuberculosis Research and Epidemiology, Mycobacterium research and diagnosis, Cytokine Signaling Pathways and Interactions, and interferon and immune responses.
h-index
36
citations
5,046
works
110
NIH funding
primary concept
email

Recent publications

Preclinical murine models for the testing of antimicrobials against Mycobacterium abscessus pulmonary infections: Current practices and recommendations
Tuberculosis 2024cited by 22position: middledoi
B cells in perivascular and peribronchiolar granuloma-associated lymphoid tissue and B-cell signatures identify asymptomatic <i>Mycobacterium tuberculosis</i> lung infection in Diversity Outbred mice
Infection and Immunity 2024cited by 13position: middledoi
Systems genetics uncover new loci containing functional gene candidates in Mycobacterium tuberculosis-infected Diversity Outbred mice
PLoS Pathogens 2024cited by 9position: middledoi
BACH1 promotes tissue necrosis and Mycobacterium tuberculosis susceptibility
Nature Microbiology 2023cited by 47position: middledoi
Cell state transition analysis identifies interventions that improve control of <i>Mycobacterium tuberculosis</i> infection by susceptible macrophages
Science Advances 2023cited by 12position: lastdoi
Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons
eLife 2021cited by 101position: middledoi
CXCL1: A new diagnostic biomarker for human tuberculosis discovered using Diversity Outbred mice
PLoS Pathogens 2021cited by 48position: middledoi
Visualizing the dynamics of tuberculosis pathology using molecular imaging
Journal of Clinical Investigation 2021cited by 31position: middledoi
Channeling macrophage polarization by rocaglates increases macrophage resistance to Mycobacterium tuberculosis
iScience 2021cited by 25position: lastdoi
The Diversity Outbred Mouse Population Is an Improved Animal Model of Vaccination against Tuberculosis That Reflects Heterogeneity of Protection
mSphere 2020cited by 61position: middledoi
The integrated stress response mediates necrosis in murine Mycobacterium tuberculosis granulomas
Journal of Clinical Investigation 2020cited by 51position: lastdoi
Type I interferon-driven susceptibility to Mycobacterium tuberculosis is mediated by IL-1Ra
Nature Microbiology 2019cited by 191position: middledoi
Spatial and temporal localization of immune transcripts defines hallmarks and diversity in the tuberculosis granuloma
Nature Communications 2019cited by 106position: middledoi
SP110b Controls Host Immunity and Susceptibility to Tuberculosis
American Journal of Respiratory and Critical Care Medicine 2017cited by 45position: middledoi
Capacity of Pneumococci to Activate Macrophage Nuclear Factor κB: Influence on Necroptosis and Pneumonia Severity
The Journal of Infectious Diseases 2017cited by 24position: middledoi
New Genome-Wide Algorithm Identifies Novel In-Vivo Expressed Mycobacterium Tuberculosis Antigens Inducing Human T-Cell Responses with Classical and Unconventional Cytokine Profiles
Scientific Reports 2016cited by 64position: middledoi
Mouse models of human TB pathology: roles in the analysis of necrosis and the development of host-directed therapies
Seminars in Immunopathology 2015cited by 175position: firstdoi
Lung necrosis and neutrophils reflect common pathways of susceptibility to<i>Mycobacterium tuberculosis</i>in genetically diverse, immune competent mice
Disease Models & Mechanisms 2015cited by 121position: middledoi
Limited Activity of Clofazimine as a Single Drug in a Mouse Model of Tuberculosis Exhibiting Caseous Necrotic Granulomas
Antimicrobial Agents and Chemotherapy 2014cited by 115position: middledoi
The sst1 Resistance Locus Regulates Evasion of Type I Interferon Signaling by Chlamydia pneumoniae as a Disease Tolerance Mechanism
PLoS Pathogens 2013cited by 33position: middledoi
Evaluation of a Mouse Model of Necrotic Granuloma Formation Using C3HeB/FeJ Mice for Testing of Drugs against Mycobacterium tuberculosis
Antimicrobial Agents and Chemotherapy 2012cited by 273position: middledoi
Autophagy Protein Rubicon Mediates Phagocytic NADPH Oxidase Activation in Response to Microbial Infection or TLR Stimulation
Cell Host & Microbe 2012cited by 155position: middledoi
Identification of regulators of the innate immune response to cytosolic DNA and retroviral infection by an integrative approach
Nature Immunology 2012cited by 133position: middledoi
The Autophagy Regulator Rubicon Is a Feedback Inhibitor of CARD9-Mediated Host Innate Immunity
Cell Host & Microbe 2012cited by 106position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Gillian Beamer · Tufts University6 papers (2015–2024)Bülent Yener · Rensselaer Polytechnic Institute4 papers (2015–2024)Metin N. Gürcan · Wake Forest University4 papers (2015–2024)Daniel M. Gatti · Jackson Laboratory4 papers (2015–2024)Melanie Ginese · Tufts University3 papers (2021–2024)Bidisha Bhattacharya · Mohammad Ali Jauhar University3 papers (2020–2023)Adam C. Gower · Boston University3 papers (2021–2024)Muhammad Khalid Khan Niazi · Wake Forest University3 papers (2015–2024)Thomas E. Tavolara · Wake Forest University3 papers (2021–2024)Deniz Koyuncu · Rensselaer Polytechnic Institute3 papers (2021–2024)Sujoy Chatterjee · Boston University3 papers (2020–2023)Anne J. Lenaerts · Colorado State University2 papers (2012–2014)Chul‐Su Yang · National Taiwan University2 papers (2012–2012)Lester Kobzik · Io Therapeutics (United States)2 papers (2020–2021) · 2 papers (2020–2024)Claudia Abeijón · Tufts University2 papers (2015–2021)Russell E. Vance · Howard Hughes Medical Institute2 papers (2019–2021)Alexander R. Ivanov · University of Massachusetts Chan Medical School2 papers (2020–2021)Jae U. Jung · Cleveland Clinic2 papers (2012–2012)Daisy X. Ji · University of California, Berkeley2 papers (2019–2021)