Area of research
Cardiology and Cardiovascular Medicine · Physiology
Research interest
Research interests include Medicine, Internal medicine, Heart failure, Cardiology, Mitochondrion, and Neprilysin.
Tachycardiomyopathy entails a dysfunctional pattern of interrelated mitochondrial functions
Travelling with heart failure: risk assessment and practical recommendations
Abstract 16339: Mitochondrial Function in Animal and Novel Human Disease Models of Tachycardiomyopathy
Skeletal muscle alterations in tachycardia-induced heart failure are linked to deficient natriuretic peptide signalling and are attenuated by RAS-/NEP-inhibition
Combined Inhibition of the Renin-Angiotensin System and Neprilysin Positively Influences Complex Mitochondrial Adaptations in Progressive Experimental Heart Failure
NT-proBNP Predicts Cardiovascular Death in the General Population Independent of Left Ventricular Mass and Function: Insights from a Large Population-Based Study with Long-Term Follow-Up
Prevalence of Sleep-Disordered Breathing-Related Symptoms in Patients with Chronic Heart Failure and Reduced Ejection Fraction
Interatrial Differences of Basal Molecular Set-Up and Changes in Tachycardia-Induced Heart Failure–A Proteomic Profiling Study
Experimental Heart Failure Induces Alterations of the Lung Proteome - Insight into Molecular Mechanisms
P654Left and right atria show different basal expression patterns of metabolic enzymes in a proteomic-based gene ontology representation study
Antidiabetic treatment restores adiponectin serum levels and APPL1 expression, but does not improve adiponectin-induced vasodilation and endothelial dysfunction in Zucker diabetic fatty rats
Expression of fourteen novel obesity-related genes in zucker diabetic fatty rats
Proteomic Profiling Implies Mitochondrial Dysfunction in Tachycardia-Induced Heart Failure
Antihypertrophic effects of combined inhibition of the renin–angiotensin system (RAS) and neutral endopeptidase (NEP) in progressive, tachycardia-induced experimental heart failure
Complete recovery after near-fatal multifocal embolization of giant cardiac thrombus: importance of rapid cerebrovascular intervention