Area of research
Physiology · Biochemistry
Research interest
Research interests include Nitric Oxide and Endothelin Effects, Antioxidant Activity and Oxidative Stress, Adipokines, Inflammation, and Metabolic Diseases, and Vitamin C and Antioxidants Research.
Mitigation of aircraft noise-induced vascular dysfunction and oxidative stress by exercise, fasting, and pharmacological α1AMPK activation: molecular proof of a protective key role of endothelial α1AMPK against environmental noise exposure
Rapid, accurate publication and dissemination of clinical trial results: benefits and challenges.
Extracorporeal CPR in Out-of-Hospital Cardiac Arrest — Still on Life Support?
Bempedoic Acid and the Prevention of Cardiovascular Disease.
Extracorporeal CPR in Out-of-Hospital Cardiac Arrest - Still on Life Support?
Association of Thromboxane Generation With Survival in Aspirin Users and Nonusers
PGC1α Regulates the Endothelial Response to Fluid Shear Stress via Telomerase Reverse Transcriptase Control of Heme Oxygenase-1.
Resternotomy Coronary Artery Bypass 1999-2018: Insights From The Society of Thoracic Surgeons Adult Cardiac Surgery Database
Cigarette Smoking Is Related to Endothelial Dysfunction of Resistance, but Not Conduit Arteries in the General Population—Results From the Gutenberg Health Study
Lack of Endothelial α1AMPK Reverses the Vascular Protective Effects of Exercise by Causing eNOS Uncoupling
Effects of tobacco cigarettes, e-cigarettes, and waterpipe smoking on endothelial function and clinical outcomes
Effects of tobacco cigarettes, e-cigarettes, and waterpipe smoking on endothelial function and clinical outcomes.
Short-term e-cigarette vapour exposure causes vascular oxidative stress and dysfunction: evidence for a close connection to brain damage and a key role of the phagocytic NADPH oxidase (NOX-2)
Short-term e-cigarette vapour exposure causes vascular oxidative stress and dysfunction: evidence for a close connection to brain damage and a key role of the phagocytic NADPH oxidase (NOX-2)
Endothelial α1AMPK modulates angiotensin II-mediated vascular inflammation and dysfunction
Endothelial α1AMPK modulates angiotensin II-mediated vascular inflammation and dysfunction.
Heart rate, mortality, and the relation with clinical and subclinical cardiovascular diseases: results from the Gutenberg Health Study
JNK and cardiometabolic dysfunction.
Neural JNK3 regulates blood flow recovery after hindlimb ischemia in mice via an Egr1/Creb1 axis
Mitral Valve Repair Versus Replacement in Elderly With Degenerative Disease: Analysis of the STS Adult Cardiac Surgery Database.
Heart rate, mortality, and the relation with clinical and subclinical cardiovascular diseases: results from the Gutenberg Health Study.
Short-term exposure to ambient air pollution and circulating biomarkers of endothelial cell activation: The Framingham Heart Study
Exercise Rescues Gene Pathways Involved in Vascular Expansion and Promotes Functional Angiogenesis in Subcutaneous White Adipose Tissue.
VITAL Signs for Dietary Supplementation to Prevent Cancer and Heart Disease.
Short-term exposure to ambient air pollution and circulating biomarkers of endothelial cell activation: The Framingham Heart Study
α1AMPK deletion in myelomonocytic cells induces a pro-inflammatory phenotype and enhances angiotensin II-induced vascular dysfunction
Short-Term Exposure to Ambient Air Pollution and Biomarkers of Systemic Inflammation
Prospective Relation of Circulating Adipokines to Incident Metabolic Syndrome: The Framingham Heart Study
Short‐Term Exposure to Air Pollution and Biomarkers of Oxidative Stress: The Framingham Heart Study
PGC-1α dictates endothelial function through regulation of eNOS expression