Area of research
Public Health, Environmental and Occupational Health · Biochemistry
Research interest
Research interests include Mosquito-borne diseases and control, Lipid metabolism and biosynthesis, COVID-19 Clinical Research Studies, and Immune Response and Inflammation.
Heme and hemozoin induce platelet cell death through UPR-induced apoptosis and ferroptosis in vivax malaria.
Platelet-neutrophil aggregate formation induces NLRP3 inflammasome activation in vaccine-induced thrombotic thrombocytopenia
IL-1β enhances susceptibility to atrial fibrillation in mice by acting through resident macrophages and promoting caspase-1 expression.
Bosutinib mitigates inflammation in experimental sepsis.
Heme and hemozoin induce platelet cell death through UPR-induced apoptosis and ferroptosis in vivax malaria
Prevention of lipid droplet accumulation by DGAT1 inhibition ameliorates sepsis-induced liver injury and inflammation.
Accumulation of lipid droplets induced by Listeria monocytogenes in macrophages: implications for survival and evasion of innate immunity.
Genetic variants in <i>DBC1</i>, <i>SIRT1</i>, <i>UCP2</i> and <i>ADRB2</i> as potential biomarkers for severe obesity and metabolic complications.
Platelet-neutrophil aggregate formation induces NLRP3 inflammasome activation in vaccine-induced thrombotic thrombocytopenia
Unlocking secrets: lipid metabolism and lipid droplet crucial roles in SARS-CoV-2 infection and the immune response.
Extracellular vesicles from primary human macrophages stimulated with VIP or PACAP mediate anti-SARS-CoV-2 activities in monocytes through NF-κB signaling pathway
Newly Proposed Dose of Daclatasvir to Prevent Lethal SARS-CoV-2 Infection in Human Transgenic ACE-2 Mice.
Microglial NLRP3 Inflammasome Induces Excitatory Synaptic Loss Through IL-1β-Enriched Microvesicle Release: Implications for Sepsis-Associated Encephalopathy.
Inhibition of the SREBP pathway prevents SARS-CoV-2 replication and inflammasome activation.
Acute to post-acute COVID-19 thromboinflammation persistence: Mechanisms and potential consequences.
Preclinical development of kinetin as a safe error-prone SARS-CoV-2 antiviral able to attenuate virus-induced inflammation.
TF/PAR2 Signaling Axis Supports the Protumor Effect of Neutrophil Extracellular Traps (NETs) on Human Breast Cancer Cells.
Lipid droplets in Zika neuroinfection: Potential targets for intervention?
The role of NSP6 in the biogenesis of the SARS-CoV-2 replication organelle.
Platelet-monocyte interaction amplifies thromboinflammation through tissue factor signaling in COVID-19.
Combination of antiviral drugs inhibits SARS-CoV-2 polymerase and exonuclease and demonstrates COVID-19 therapeutic potential in viral cell culture.
Simvastatin Downregulates the SARS-CoV-2-Induced Inflammatory Response and Impairs Viral Infection Through Disruption of Lipid Rafts.
Persisting Platelet Activation and Hyperactivity in COVID-19 Survivors.
Commercially Available Flavonols Are Better SARS-CoV-2 Inhibitors than Isoflavone and Flavones.
Human endogenous retrovirus K in the respiratory tract is associated with COVID-19 physiopathology.
Platelet proteome reveals features of cell death, antiviral response and viral replication in covid-19.
Lipid droplet accumulation occurs early following Salmonella infection and contributes to intracellular bacterial survival and replication.
Platelet-leukocyte interactions in the pathogenesis of viral infections.
VIP plasma levels associate with survival in severe COVID-19 patients, correlating with protective effects in SARS-CoV-2-infected cells.
SARS-CoV-2: Ultrastructural Characterization of Morphogenesis in an In Vitro System.