Area of research
Immunology · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Medicine, Internal medicine, Inflammation, Myocardial infarction, NADPH oxidase, and Cardiology.
Induction of a distinct macrophage population and protection from lung injury and fibrosis by Notch2 blockade
Macrophage-Expressed Coagulation Factor VII Promotes Adverse Cardiac Remodeling
Myeloid cell-derived interleukin-6 induces vascular dysfunction and vascular and systemic inflammation
Macrophage-expressed coagulation factor 7 promotes adverse cardiac remodeling
Aircraft noise exposure induces pro-inflammatory vascular conditioning and amplifies vascular dysfunction and impairment of cardiac function after myocardial infarction
Telomere Shortening in Hypertensive Heart Disease Depends on Oxidative DNA Damage and Predicts Impaired Recovery of Cardiac Function in Heart Failure
Targeting myeloid cell coagulation signaling blocks MAP kinase/TGF-β1–driven fibrotic remodeling in ischemic heart failure
<i>Aestivation</i>motifs explain hypertension and muscle mass loss in mice with psoriatic skin barrier defect
ACE Inhibition Modulates Myeloid Hematopoiesis after Acute Myocardial Infarction and Reduces Cardiac and Vascular Inflammation in Ischemic Heart Failure
Nox2+ myeloid cells drive vascular inflammation and endothelial dysfunction in heart failure after myocardial infarction via angiotensin II receptor type 1
Angiotensin II Infusion Leads to Aortic Dissection in LRP8 Deficient Mice
T Cell-Derived IL-17A Induces Vascular Dysfunction via Perivascular Fibrosis Formation and Dysregulation of<sup>⋅</sup>NO/cGMP Signaling
A sequential interferon gamma directed chemotactic cellular immune response determines survival and cardiac function post-myocardial infarction
Pulmonary Arterial Hypertension and Endothelial Dysfunction Is Linked to NADPH Oxidase‐Derived Superoxide Formation in Venous Thrombosis and Pulmonary Embolism in Mice
Hypoxia evokes increased PDI and PDIA6 expression in the infarcted myocardium of ex-germ-free and conventionally-raised mice
NOX2 amplifies acetaldehyde-mediated cardiomyocyte mitochondrial dysfunction in alcoholic cardiomyopathy