Michael E. DeBakey VA Medical Center · US
Area of research
Biomedical Engineering · Surgery
Research interest
Research interests include Mechanical Circulatory Support Devices, Cardiac Structural Anomalies and Repair, Cardiovascular Function and Risk Factors, and Cardiac Valve Diseases and Treatments.
Noninvasive Pulmonary Capillary Wedge Pressure Estimation in Heart Failure Patients With the Use of Wearable Sensing and AI
The RNA editor ADAR2 promotes immune cell trafficking by enhancing endothelial responses to interleukin-6 during sterile inflammation
State‐of‐the‐art machine learning algorithms for the prediction of outcomes after contemporary heart transplantation: Results from the UNOS database
Circulating and Myocardial Cytokines Predict Cardiac Structural and Functional Improvement in Patients With Heart Failure Undergoing Mechanical Circulatory Support
The pyruvate-lactate axis modulates cardiac hypertrophy and heart failure
Insulin receptor substrates differentially exacerbate insulin-mediated left ventricular remodeling
Genome-wide DNA methylation encodes cardiac transcriptional reprogramming in human ischemic heart failure
Clinical and Histopathological Effects of Heart Failure Drug Therapy in Advanced Heart Failure Patients on Chronic Mechanical Circulatory Support
Evidence of Glycolysis Up-Regulation and Pyruvate Mitochondrial Oxidation Mismatch During Mechanical Unloading of the Failing Human Heart
Impact of Ischemic Heart Failure Etiology on Cardiac Recovery During Mechanical Unloading
EVIDENCE OF GLYCOLYSIS UPREGULATION AND PYRUVATE MITOCHONDRIAL OXIDATION MISMATCH DURING MECHANICAL UNLOADING OF THE FAILING HUMAN HEART: IMPLICATIONS FOR CARDIAC RELOADING AND CONDITIONING
Cardiac, skeletal, and smooth muscle mitochondrial respiration: are all mitochondria created equal?
Myocardial Atrophy and Chronic Mechanical Unloading of the Failing Human Heart
Characterization of diffuse fibrosis in the failing human heart via diffusion tensor imaging and quantitative histological validation
Magnitude and Time Course of Changes Induced by Continuous-Flow Left Ventricular Assist Device Unloading in Chronic Heart Failure