Area of research
Infectious Diseases · Molecular Biology
Research interest
Research interests include Biology, Candida albicans, DNA damage, Gene, Hepatic stellate cell, and Cell biology.
DNA damage repair factor Rad18 controls virulence partially via transcriptional suppression of genes <i>HWP1</i> and <i>ECE1</i> in <i>Candida albicans</i>
Amino acid metabolism and MAP kinase signaling pathway play opposite roles in the regulation of ethanol production during fermentation of sugarcane molasses in budding yeast
Loss of Gst1 enhances resistance to MMS by reprogramming the transcription of DNA damage response genes in a Rad53-dependent manner in Candida albicans
Transcriptional Profiling of the Candida albicans Response to the DNA Damage Agent Methyl Methanesulfonate
Modulation of the complex regulatory network for methionine biosynthesis in fungi
DNA damage checkpoint and repair: From the budding yeast <i>Saccharomyces cerevisiae</i> to the pathogenic fungus <i>Candida albicans</i>
The zinc cluster transcription factor Rha1 is a positive filamentation regulator in <i>Candida albicans</i>
Nucleotide Excision Repair Protein Rad23 Regulates Cell Virulence Independent of Rad4 in Candida albicans
Hof1 plays a checkpoint-related role in MMS-induced DNA damage response in <i>Candida albicans</i>
Inhibition of microRNA-155 modulates endotoxin tolerance by upregulating suppressor of cytokine signaling 1 in microglia
A label-free and ultrasensitive electrochemical aptasensor for lead(<scp>ii</scp>) using a N,P dual-doped carbon dot–chitosan composite as a signal-enhancing platform and thionine as a signaling molecule
The N-terminal pY33XL motif of CaPsy2 is critical for the function of protein phosphatase 4 in CaRad53 deactivation, DNA damage-induced filamentation and virulence in Candida albicans
Lipopolysaccharide Preconditioning Induces an Anti-inflammatory Phenotype in BV2 Microglia
CaTip41 regulates protein phosphatase 2A activity, CaRad53 deactivation and the recovery of DNA damage-induced filamentation to yeast form in<i>Candida albicans</i>
Macrophage-Microglia Networks Drive M1 Microglia Polarization After Mycobacterium Infection
Expression of microRNA-454 in TGF-β1-stimulated hepatic stellate cells and in mouse livers infected with Schistosoma japonicum
Schistosoma japonicum soluble egg antigens induce apoptosis and inhibit activation of hepatic stellate cells: a possible molecular mechanism
Schistosoma japonicum Soluble Egg Antigens Facilitate Hepatic Stellate Cell Apoptosis by Downregulating Akt Expression and Upregulating p53 and DR5 Expression
Mithochondrial type 2C protein phosphatases CaPtc5p, CaPtc6p, and CaPtc7p play vital roles in cellular responses to antifungal drugs and cadmium in<i>Candida albicans</i>
Genetic interactions between protein phosphatases CaPtc2p and CaPph3p in response to genotoxins and rapamycin in<i>Candida albicans</i>