Area of research
Oncology · Immunology
Research interest
Research interests include Immunology, Medicine, Chemokine, Biology, Inflammation, and Cell biology.
Syntenin-1-mediated arthritogenicity is advanced by reprogramming RA metabolic macrophages and Th1 cells
Interleukin‐34 Reprograms Glycolytic and Osteoclastic Rheumatoid Arthritis Macrophages via Syndecan 1 and Macrophage Colony‐Stimulating Factor Receptor
Metabolic reprogramming of macrophages instigates CCL21‐induced arthritis
IRAK4 inhibition: a promising strategy for treating RA joint inflammation and bone erosion
The pathogenic importance of CCL21 and CCR7 in rheumatoid arthritis
TLR7 endogenous ligands remodel glycolytic macrophages and trigger skin‐to‐joint crosstalk in psoriatic arthritis
CCL25 and CCR9 is a unique pathway that potentiates pannus formation by remodeling RA macrophages into mature osteoclasts
CCL21/CCR7 signaling in macrophages promotes joint inflammation and Th17-mediated osteoclast formation in rheumatoid arthritis
Macrophages are the primary effector cells in IL-7-induced arthritis
IL-11 facilitates a novel connection between RA joint fibroblasts and endothelial cells
Impact of obesity on autoimmune arthritis and its cardiovascular complications
Recombinant Parvoviruses Armed to Deliver CXCL4L1 and CXCL10 Are Impaired in Their Antiangiogenic and Antitumoral Effects in a Kaposi Sarcoma Tumor Model Due To the Chemokines' Interference with the Virus Cycle
CXCL4 and CXCL4L1 Differentially Affect Monocyte Survival and Dendritic Cell Differentiation and Phagocytosis
Myofibroblasts in proliferative diabetic retinopathy can originate from infiltrating fibrocytes and through endothelial-to-mesenchymal transition (EndoMT)
CXCR3 ligands in disease and therapy
Autocrine CCL2, CXCL4, CXCL9 and CXCL10 signal in retinal endothelial cells and are enhanced in diabetic retinopathy