Area of research
Clinical Biochemistry · Rehabilitation
Research interest
Research interests include Rage (emotion), Biology, SOD1, Motor neuron, Endocrinology, and Amyotrophic lateral sclerosis.
Predicting disease specific spinal motor neurons and glia in sporadic ALS
RAGE binds preamyloid IAPP intermediates and mediates pancreatic β cell proteotoxicity
Mutant SOD1 prevents normal functional recovery through enhanced glial activation and loss of motor neuron innervation after peripheral nerve injury
<i>Ager</i> Deletion Enhances Ischemic Muscle Inflammation, Angiogenesis, and Blood Flow Recovery in Diabetic Mice
Slowing disease progression in the SOD1 mouse model of ALS by blocking neuregulin-induced microglial activation
Neuregulin1 fine‐tunes pre‐, post‐, and perisynaptic neuromuscular junction development
RAGE Suppresses ABCG1-Mediated Macrophage Cholesterol Efflux in Diabetes
RAGE Regulates the Metabolic and Inflammatory Response to High-Fat Feeding in Mice
Predicting novel histopathological microlesions in human epileptic brain through transcriptional clustering
Activation of microglial neuregulin1 signaling in the corticospinal tracts of ALS patients with upper motor neuron signs
Lysophosphatidic acid targets vascular and oncogenic pathways via RAGE signaling
Aberrant Neuregulin 1 Signaling in Amyotrophic Lateral Sclerosis
Glycation and Insulin Resistance