Area of research
Hepatology · Epidemiology
Research interest
Research interests include Hepatitis C virus research, Hepatitis B Virus Studies, Liver Disease Diagnosis and Treatment, and interferon and immune responses.
Taxonomic expansion and reorganization of Flaviviridae
Reconstitution of interferon regulatory factor 7 expression restores interferon beta induction in Huh7 cells
RBM39 shapes innate immunity by controlling the expression of key factors of the interferon response
Phosphatidylinositol 4-Kinase III Alpha Governs Cytoskeletal Organization for Invasiveness of Liver Cancer Cells
Author Reply to Peer Reviews of RBM39 shapes innate immunity through transcriptional and splicing control of key factors of the interferon response
Renaming of the genus Flavivirus to Orthoflavivirus and extension of binomial species names within the family Flaviviridae
Comparison of HAV and HCV infections in vivo and in vitro reveals distinct patterns of innate immune evasion and activation
Extended methods for spatial cell classification with DBSCAN-CellX
Joint statement in support of hepatitis C human challenge studies
RBM39 shapes innate immunity through transcriptional and splicing control of key factors of the interferon response
A Hepatitis C virus genotype 1b post-transplant isolate with high replication efficiency in cell culture and its adaptation to infectious virus production in vitro and in vivo
A Hepatitis C virus genotype 1b post-transplant isolate with high replication efficiency in cell culture and its adaptation to infectious virus production in vitro and in vivo
Comparison of HAV and HCV infections <i>in vivo</i> and <i>in vitro</i> reveals distinct patterns of innate immune evasion and activation
Convergent use of phosphatidic acid for hepatitis C virus and SARS-CoV-2 replication organelle formation
Convergent use of phosphatidic acid for Hepatitis C virus and SARS-CoV-2 replication organelle formation
Indelibly Stamped by Hepatitis C Virus Infection: Persistent Epigenetic Signatures Increasing Liver Cancer Risk
Evidence for Internal Initiation of RNA Synthesis by the Hepatitis C Virus RNA-Dependent RNA Polymerase NS5B <i>In Cellulo</i>
The Hepatitis C Virus Replicon System and Its Role in Drug Development
Critical challenges and emerging opportunities in hepatitis C virus research in an era of potent antiviral therapy: Considerations for scientists and funding agencies
microRNA-122 amplifies hepatitis C virus translation by shaping the structure of the internal ribosomal entry site
Secretion of Hepatitis C Virus Replication Intermediates Reduces Activation of Toll-Like Receptor 3 in Hepatocytes
Characterization of a Threonine-Rich Cluster in Hepatitis C Virus Nonstructural Protein 5A and Its Contribution to Hyperphosphorylation
Flavivirus Infection Uncouples Translation Suppression from Cellular Stress Responses
Phosphorylation-Dependent Feedback Inhibition of RIG-I by DAPK1 Identified by Kinome-wide siRNA Screening
Overall Structural Model of NS5A Protein from Hepatitis C Virus and Modulation by Mutations Confering Resistance of Virus Replication to Cyclosporin A
Erratum for Roth et al., “Flavivirus Infection Uncouples Translation Suppression from Cellular Stress Responses”
Hepatitis C virus-induced natural killer cell proliferation involves monocyte-derived cells and the OX40/OX40L axis
Mutations in Encephalomyocarditis Virus 3A Protein Uncouple the Dependency of Genome Replication on Host Factors Phosphatidylinositol 4-Kinase IIIα and Oxysterol-Binding Protein
Tuning a cellular lipid kinase activity adapts hepatitis C virus to replication in cell culture
Modulation of the Host Lipid Landscape to Promote RNA Virus Replication: The Picornavirus Encephalomyocarditis Virus Converges on the Pathway Used by Hepatitis C Virus