Area of research
Molecular Biology · Ophthalmology
Research interest
Research interests include Connexins and lens biology, Intraocular Surgery and Lenses, Autophagy in Disease and Therapy, and Neuroinflammation and Neurodegeneration Mechanisms.
Carbon source-driven downregulation of the sRNA AmiL enhances <i>Pseudomonas aeruginosa</i> virulence during acute lung infection
Global prevalence, trend and projection of myopia in children and adolescents from 1990 to 2050: a comprehensive systematic review and meta-analysis
Insights into the molecular mechanisms underlying the different heat tolerance of the scleractinian coral Pavona decussata
Metabolic signatures of two scleractinian corals from the northern South China sea in response to extreme high temperature events
The lens epithelium as a major determinant in the development, maintenance, and regeneration of the crystalline lens
Increases in miR-124-3p in Microglial Exosomes Confer Neuroprotective Effects by Targeting FIP200-Mediated Neuronal Autophagy Following Traumatic Brain Injury
Characterization of Exosomes in Plasma of Patients with Breast, Ovarian, Prostate, Hepatic, Gastric, Colon, and Pancreatic Cancers
Valosin‑containing protein promotes metastasis of osteosarcoma through autophagy induction and anoikis inhibition via the ERK/NF‑κβ/beclin‑1 signaling pathway
The pathological role of NLRs and AIM2 inflammasome-mediated pyroptosis in damaged blood-brain barrier after traumatic brain injury
Transplantation of in vitro cultured endothelial progenitor cells repairs the blood-brain barrier and improves cognitive function of APP/PS1 transgenic AD mice
Increased miR‐124‐3p in microglial exosomes following traumatic brain injury inhibits neuronal inflammation and contributes to neurite outgrowth <i>via</i> their transfer into neurons
Flow Cytometric Characterization of T Cell Subsets and Microglia After Repetitive Mild Traumatic Brain Injury in Rats
Lens regeneration using endogenous stem cells with gain of visual function
Knockdown of Aurora-B inhibits osteosarcoma cell invasion and migration via modulating PI3K/Akt/NF-κB signaling pathway.
PubMed 2014cited by 43position: last
Inhibition of Aurora-B suppresses osteosarcoma cell migration and invasion
Let-7g reverses malignant phenotype of osteosarcoma cells by targeting Aurora-B.
PubMed 2014cited by 19position: middle
RNA interference-mediated knockdown of Aurora-B alters the metastatic behavior of A549 cells via modulation of the phosphoinositide 3-kinase/Akt signaling pathway