Area of research
Molecular Biology · Nephrology
Research interest
Research interests include Mitochondrion, Cell biology, Endoplasmic reticulum, Medicine, Uniporter, and Heart failure.
Mechano-energetic uncoupling in heart failure
Opportunities and challenges for the use of human samples in translational cardiovascular research: a scientific statement of the ESC Working Group on Cellular Biology of the Heart, the ESC Working Group on Cardiovascular Surgery, the ESC Council on Basic Cardiovascular Science, the ESC Scientists of Tomorrow, the European Association of Percutaneous Cardiovascular Interventions of the ESC, and the Heart Failure Association of the ESC
Mitochondrial Targets in Ischaemic Heart Disease and Heart Failure, and their Potential for a More Efficient Clinical Translation. A Scientific Statement of the ESC Working Group on Cellular Biology of the Heart and the ESC Working Group on Myocardial Function
Myocardial transcriptomic analysis of diabetic patients with aortic stenosis: key role for mitochondrial calcium signaling
SERCA2 phosphorylation at serine 663 is a key regulator of Ca2+ homeostasis in heart diseases
Long COVID and the cardiovascular system—elucidating causes and cellular mechanisms in order to develop targeted diagnostic and therapeutic strategies: a joint Scientific Statement of the ESC Working Groups on Cellular Biology of the Heart and Myocardial and Pericardial Diseases
Endoplasmic reticulum-mitochondria miscommunication is an early and causal trigger of hepatic insulin resistance and steatosis
Altered composition of the mitochondrial Ca2+uniporter in the failing human heart
Reduced reticulum–mitochondria Ca2+ transfer is an early and reversible trigger of mitochondrial dysfunctions in diabetic cardiomyopathy
Pharmacological inhibition of the mitochondrial Ca2+ uniporter: Relevance for pathophysiology and human therapy
PET-MRI nanoparticles imaging of blood–brain barrier damage and modulation after stroke reperfusion
A novel class of cardioprotective small-molecule PTP inhibitors
Mitochondrial fusion dynamics is robust in the heart and depends on calcium oscillations and contractile activity
Disruption of calcium transfer from ER to mitochondria links alterations of mitochondria-associated ER membrane integrity to hepatic insulin resistance
The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury
Depressing Mitochondria-Reticulum Interactions Protects Cardiomyocytes From Lethal Hypoxia-Reoxygenation Injury