Area of research
Epidemiology · Molecular Biology
Research interest
Research interests include Adipose Tissue and Metabolism, Liver Disease Diagnosis and Treatment, Adipokines, Inflammation, and Metabolic Diseases, and Melanoma and MAPK Pathways.
Microglia Rank signaling regulates GnRH neuronal function and the hypothalamic-pituitary-gonadal axis.
TUMOR–PRE-ADIPOCYTE CROSSTALK SUSTAINS BREAST CANCER GROWTH VIA RET SIGNALLING
The aging modulator miR-29 is essential for adult cardiomyocyte function
A hypothalamus-liver-skeletal muscle axis controlled by JNK1 and FGF21 mediates olanzapine-induced insulin resistance in an intraperitoneal treatment in male mice
Inhibition of p107 alleviates liver steatosis by reducing de novo fatty acid synthesis
Absence of MCJ/DnaJC15 promotes brown adipose tissue thermogenesis
Absence of MCJ/DnaJC15 promotes brown adipose tissue thermogenesis.
Cx43 enhances response to BRAF/MEK inhibitors by reducing DNA repair capacity.
Adipocyte MKK3 Increases in Human Obesity protecting against Insulin Resistance by p38β activation
Author Reply to Peer Reviews of Loss of the alternative calcineurin variant CnAβ1 enhances brown adipocyte differentiation and drives metabolic overactivation through FoxO1 activation
Loss of the alternative calcineurin variant CnAβ1 enhances brown adipocyte differentiation and drives metabolic overactivation through FoxO1 activation
Remodeling p38 signaling in muscle controls locomotor activity via IL-15.
MCJ: A mitochondrial target for cardiac intervention in pulmonary hypertension
From Beats to Metabolism: the Heart at the Core of Interorgan Metabolic Cross Talk.
MCJ: A mitochondrial target for cardiac intervention in pulmonary hypertension.
Hepatic nutrient and hormone signaling to mTORC1 instructs the postnatal metabolic zonation of the liver.
DIDO is necessary for the adipogenesis that promotes diet-induced obesity
Lack of p38 activation in T cells increases IL-35 and protects against obesity by promoting thermogenesis.
p38α kinase governs muscle strength through PGC1α in mice.
The outcome of boosting mitochondrial activity in alcohol-associated liver disease is organ-dependent.
Myeloid p38 activation maintains macrophage-liver crosstalk and BAT thermogenesis through IL-12-FGF21 axis.
Predominantly Pro-Inflammatory Phenotype with Mixed M1/M2 Polarization of Peripheral Blood Classical Monocytes and Monocyte-Derived Macrophages among Patients with Excessive Ethanol Intake.
Hepatic stellate cell activation markers are regulated by the vagus nerve in systemic inflammation.
Hypothalamic JNK1-hepatic fatty acid synthase axis mediates a metabolic rewiring that prevents hepatic steatosis in male mice treated with olanzapine via intraperitoneal: Additional effects of PTP1B inhibition.
p38γ/δ activation alters cardiac electrical activity and predisposes to ventricular arrhythmia.
Lack of p38 activation in T cells increases IL-35 production and protects against obesity by promoting thermogenesis
Prolonged breastfeeding protects from obesity by hypothalamic action of hepatic FGF21.
Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals.
Inhibition of ATG3 ameliorates liver steatosis by increasing mitochondrial function
Methionine adenosyltransferase 1a antisense oligonucleotides activate the liver-brown adipose tissue axis preventing obesity and associated hepatosteatosis.