Area of research
Pulmonary and Respiratory Medicine · Immunology
Research interest
Research interests include Chronic Obstructive Pulmonary Disease (COPD) Research, Immune Cell Function and Interaction, Hepatitis C virus research, and Respiratory Support and Mechanisms.
IL-17A-producing NKp44(−) group 3 innate lymphoid cells accumulate in Familial Adenomatous Polyposis duodenal tissue
IL-17A-producing NKp44(-) group 3 innate lymphoid cells accumulate in Familial Adenomatous Polyposis duodenal tissue.
The life-saving benefit of dexamethasone in severe COVID-19 is linked to a reversal of monocyte dysregulation
Catecholamines and inflammation in advanced liver disease: more closely intertwined than expected?
Single-cell RNA sequencing identifies a population of human liver-type ILC1s
Single-cell RNA sequencing identifies a population of human liver-type ILC1s.
IL-6-Dependent STAT3 Activation and Induction of Proinflammatory Cytokines in Primary Sclerosing Cholangitis.
Identification and characterization of a hepatic IL-13-producing ILC3-like population potentially involved in liver fibrosis.
High producer variant of lipoprotein lipase may protect from hepatocellular carcinoma in alcohol-associated cirrhosis
HIV-Associated Microbial Translocation May Affect Cytokine Production of CD56bright NK Cells via Stimulation of Monocytes.
Natural Killer Cell-Mediated Antibody-Dependent Cellular Cytotoxicity Against SARS-CoV-2 After Natural Infection Is More Potent Than After Vaccination.
Mitochondrial Dysfunction Contributes to Impaired Cytokine Production of CD56bright Natural Killer Cells From Human Immunodeficiency Virus-Infected Individuals Under Effective Antiretroviral Therapy.
Genetic Variation of <i>SAMM50</i> Is Not an Independent Risk Factor for Alcoholic Hepatocellular Carcinoma in Caucasian Patients.
Swarm Learning for decentralized and confidential clinical machine learning
Swarm Learning for decentralized and confidential clinical machine learning
Early IFN-α signatures and persistent dysfunction are distinguishing features of NK cells in severe COVID-19
Disease severity-specific neutrophil signatures in blood transcriptomes stratify COVID-19 patients.
Early IFN-α signatures and persistent dysfunction are distinguishing features of NK cells in severe COVID-19.
Severe COVID-19 Is Marked by a Dysregulated Myeloid Cell Compartment
Suppressive myeloid cells are a hallmark of severe COVID-19
Role of regulatory T cells and checkpoint inhibition in hepatocellular carcinoma.
NK Cells in Ascites From Liver Disease Patients Display a Particular Phenotype and Take Part in Antibacterial Immune Response
The PNPLA3 I148M variant promotes lipid-induced hepatocyte secretion of CXC chemokines establishing a tumorigenic milieu.
NK Cells in Ascites From Liver Disease Patients Display a Particular Phenotype and Take Part in Antibacterial Immune Response.
Angiotensin-II Type 1 Receptor-Mediated Janus Kinase 2 Activation Induces Liver Fibrosis
Effect of once‐daily indacaterol in a predominantly <scp>C</scp>hinese population with chronic obstructive pulmonary disease: A 26‐week <scp>A</scp>sia‐<scp>P</scp>acific study
Activation of Invariant NK T Cells in Periodontitis Lesions