Kelly L. Rogers

GID/CTLH E3 ligase complex control cell fate programs for sexual development of Plasmodium falciparum.

Author Correction: Age-related epithelial defects limit thymic function and regeneration.

<i>SpatialBench</i> : Comparative cross-platform benchmarking of high-resolution spatial transcriptomics using matched mouse lymphoid tissue

MMV687794 blocks <i>Plasmodium falciparum</i> invasion of red blood cells by targeting a Surface-associated Lipid-Interacting Rhoptry Protein, <i>Pf</i> SLIRP

Formation of the moving junction is the nexus for host cytoskeletal remodelling during <i>Plasmodium falciparum</i> invasion of human erythrocytes

Transient inhibition of type I interferon enhances CD8+ T cell stemness and vaccine protection.

Profiling the spatial architecture of multiple myeloma in human bone marrow trephine biopsy specimens with spatial transcriptomics.

Differentiation of <i>Toxoplasma</i> into latent forms is linked to central carbon metabolism and requires a GID/CTLH-type E3 ubiquitin ligase

Genetic barcoding uncovers the clonal makeup of solid and liquid biopsies and their ability to capture intra-tumoral heterogeneity

Expanding channels enhanced diffractive SAW actuated particle enrichment in vacuum-sealed microfluidic channels.

The formation of the 'footprint of death' as a mechanism for generating large substrate-bound extracellular vesicles that mark the site of cell death.

Author Correction: In situ visualization of endothelial cell-derived extracellular vesicle formation in steady state and malignant conditions.

Leveraging the Rich Spatiotemporal Features of Lattice Light-sheet Microscopy with Machine Learning and AI

Ex-SCAPE: Fast Volumetric Imaging of Expanded Biological Samples using Oblique Plane Light Sheet Microscopy

Age-related epithelial defects limit thymic function and regeneration.

An immunohistochemical atlas of necroptotic pathway expression

Aryl amino acetamides prevent Plasmodium falciparum ring development via targeting the lipid-transfer protein PfSTART1

Aryl amino acetamides prevent Plasmodium falciparum ring development via targeting the lipid-transfer protein PfSTART1.

In situ visualization of endothelial cell-derived extracellular vesicle formation in steady state and malignant conditions.

Spotlight on 10x Visium: a multi-sample protocol comparison of spatial technologies

Transient inhibition of type I interferon enhances CD8 ⁺ T cell stemness and vaccine protection

Co-clustering of EphB6 and ephrinB1 in trans restrains cancer cell invasion.

Author Correction: Aryl amino acetamides prevent Plasmodium falciparum ring development via targeting the lipid-transfer protein PfSTART1.

TNF⍰-mediated myeloid-instructed CD14 ⁺ CD4 ⁺ T cells within the tumor microenvironment are associated with poor survival in non-small cell lung cancer

The anti-cancer agent APR-246 can activate several programmed cell death processes to kill malignant cells.

Trabid patient mutations impede the axonal trafficking of adenomatous polyposis coli to disrupt neurite growth.

Spatial analysis of Hofbauer cell transcriptome, distribution and morphology in placentas exposed to <i>Plasmodium falciparum</i>

Correction to: The anti-cancer agent APR-246 can activate several programmed cell death processes to kill malignant cells.

Aryl amino acetamides prevent the development of <i>Plasmodium falciparum</i> rings via inhibition of the lipid transfer protein PfSTART1

Trabid patient mutations impede the axonal trafficking of adenomatous polyposis coli to disrupt neurite growth