Area of research
Molecular Biology · Cancer Research
Research interest
Research interests include Kruppel-like factors research, RNA modifications and cancer, Cancer-related molecular mechanisms research, and Aortic Disease and Treatment Approaches.
Cholesterol induced-mitochondrial calcium dysregulation facilitates atherosclerosis by promoting lipid accumulation in vascular smooth muscle cells
Multi-drug pharmacotyping improves therapy prediction in pancreatic cancer organoids
Endoscopic versus percutaneous drainage placement after left-sided pancreatectomy: multi-state model for defining objectifiable variables on interventional treatment for pancreatic fistula
A STAT1-GBP3-STING positive feedback loop governs inflammation, oxidative stress, and DNA damage to trigger acute aortic dissection
Ythdc1‐p300‐Klf5 Complex‐Mediated Golgi Dysfunction Promotes Aortic Aneurysm
Monocyte-to-High-Density Lipoprotein-Cholesterol Ratio Predicts Prognosis of Hepatocellular Carcinoma in Patients with Metabolic-Associated Fatty Liver Disease
Cytokine‐armed vaccinia virus promotes cytotoxicity toward pancreatic carcinoma cells via activation of human intermediary <scp>CD56<sup>dim</sup>CD16<sup>dim</sup></scp> natural killer cells
Combined DeRitis ratio and alkaline phosphatase on the prediction of portal vein tumor thrombosis in patients with hepatocellular carcinoma
α-HBDH is a superior to LDH in predicting major adverse cardiovascular events in patients with acute aortic dissection
Combined DeRitis ratio and alkaline phosphatase on the Prediction of Portal Vein Tumor Thrombosis in Patients with Hepatocellular Carcinoma
Combined Deritis Ratio And Alkaline Phosphatase on the Prediction Of Portal Vein Tumor Thrombosis in Patients with Hepatocellular Carcinoma
Exosomes‐transferred LINC00668 Contributes to Thrombosis by Promoting NETs Formation in Inflammatory Bowel Disease
Predictive value of admission CO2 combining power combined with serum sodium for the prognosis in acute Stanford type A aortic dissection patients
Additional file 2 of Pyroptosis patterns influence the clinical outcome and immune microenvironment characterization in HPV-positive head and neck squamous cell carcinoma
Gasdermin D Deficiency in Vascular Smooth Muscle Cells Ameliorates Abdominal Aortic Aneurysm Through Reducing Putrescine Synthesis
KLF4-PFKFB3-driven glycolysis is essential for phenotypic switching of vascular smooth muscle cells
Decomposition kinetics and postmortem production of hydrogen sulfide and its metabolites
Carbon-dioxide-combining-power and postoperative short-term mortality in patients with Stanford type A acute aortic dissection
Anxa1 in smooth muscle cells protects against acute aortic dissection
KLF5-mediated Eppk1 expression promotes cell proliferation in cervical cancer via the p38 signaling pathway
Serum sodium on admission affects postoperative in-hospital mortality in acute aortic dissection patients
Klf5 down-regulation induces vascular senescence through eIF5a depletion and mitochondrial fission
Vascular Smooth Muscle FTO Promotes Aortic Dissecting Aneurysms via m6A Modification of Klf5
p38α in macrophages aggravates arterial endothelium injury by releasing IL-6 through phosphorylating megakaryocytic leukemia 1
Hypermethylation of Single CpG Dinucleotides at the Promoter of CXCL13 Gene Promoting Cell Migration in Cervical Cancer
eIF5a reduction via decreased Klf5 leads to cell senescence by mitochondrial fission in VSMCs
Serum- and glucocorticoid-inducible kinase 3 is a potential oncogene in nasopharyngeal carcinoma
Oxidative Stress Induces Neuronal Apoptosis Through Suppressing Transcription Factor EB Phosphorylation at Ser467
KLF5 promotes cervical cancer proliferation, migration and invasion in a manner partly dependent on TNFRSF11a expression
Inhibition of KLF5–Myo9b–RhoA Pathway–Mediated Podosome Formation in Macrophages Ameliorates Abdominal Aortic Aneurysm