Area of research
Infectious Diseases · Epidemiology
Research interest
Research interests include Biology, Autophagy, Cell biology, Virology, Virus, and Infectious bursal disease.
PDPK1 regulates autophagosome biogenesis by binding to PIK3C3
The Autophagy Cargo Receptor SQSTM1 Inhibits Infectious Bursal Disease Virus Infection through Selective Autophagic Degradation of Double-Stranded Viral RNA
PDPK1 regulates autophagosome biogenesis by binding to PIK3C3
Influenza A Virus Induces Autophagy by Its Hemagglutinin Binding to Cell Surface Heat Shock Protein 90AA1
Cytoplasmic Cargo Receptor p62 Inhibits Avibirnavirus Replication by Mediating Autophagic Degradation of Viral Protein VP2
Circular RNA GATAD2A promotes H1N1 replication through inhibiting autophagy
Binding of <i>Avibirnavirus</i> VP3 to the PIK3C3-PDPK1 complex inhibits autophagy by activating the AKT-MTOR pathway
SUMO1 Modification Facilitates Avibirnavirus Replication by Stabilizing Polymerase VP1
Analysis of Expression Profiles of Long Noncoding RNAs and mRNAs in A549 Cells Infected with H3N2 Swine Influenza Virus by RNA Sequencing
Ubiquitination Is Essential for Avibirnavirus Replication by Supporting VP1 Polymerase Activity
BECN1-dependent CASP2 incomplete autophagy induction by binding to rabies virus phosphoprotein
Rabies Virus Infection Induces Microtubule Depolymerization to Facilitate Viral RNA Synthesis by Upregulating HDAC6
Characterization of H7N2 Avian Influenza Virus in Wild Birds and Pikas in Qinghai-Tibet Plateau Area
Genetic characterization of H9N2 avian influenza virus in plateau pikas in the Qinghai Lake region of China
Binding of the pathogen receptor HSP90AA1 to avibirnavirus VP2 induces autophagy by inactivating the AKT-MTOR pathway
In Vitro Coinfection and Replication of Classical Swine Fever Virus and Porcine Circovirus Type 2 in PK15 Cells
Avibirnavirus VP4 Protein Is a Phosphoprotein and Partially Contributes to the Cleavage of Intermediate Precursor VP4-VP3 Polyprotein
iTRAQ‐based quantitative subcellular proteomic analysis of <i>Avibirnavirus</i>‐infected cells
Inhibition of Antiviral Innate Immunity by Birnavirus VP3 Protein via Blockage of Viral Double-Stranded RNA Binding to the Host Cytoplasmic RNA Detector MDA5