Area of research
Genetics · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Virus-based gene therapy research, Viral Infections and Immunology Research, CAR-T cell therapy research, and Viral gastroenteritis research and epidemiology.
Data from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration.
Supplementary Figure S5 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Supplementary Table S2 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Supplementary Figure S4 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Supplementary Figure S2 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Supplementary Figure S3 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Supplementary Table S3 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Supplementary Figure S6 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Supplementary Table 1 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Viral Microglia Reprogramming Clears Oligomeric Neurotoxic Debris
Supplementary Figure S1 from Lymphotropic Virotherapy Induces DC and High Endothelial Venule Inflammation, Promoting the Antitumor Efficacy of Intratumor Virus Administration
Polio virotherapy provokes MDA5 signaling and CD4<sup>+</sup> T cell help to mediate cancer <i>in situ</i> vaccination.
Elucidating cellular response to treatment with viral immunotherapies in pediatric high-grade glioma and medulloblastoma
Polio virotherapy targets the malignant glioma myeloid infiltrate with diffuse microglia activation engulfing the CNS
Intratumor childhood vaccine-specific CD4<sup>+</sup> T-cell recall coordinates antitumor CD8<sup>+</sup> T cells and eosinophils
Early enterovirus translation deficits extend viral RNA replication and elicit sustained MDA5-directed innate signaling.
PKR Binds Enterovirus IRESs, Displaces Host Translation Factors, and Impairs Viral Translation to Enable Innate Antiviral Signaling.
CD155 is a putative therapeutic target in medulloblastoma
Polio Virotherapy of Malignant Glioma Engages the Tumor Myeloid Infiltrate and Induces Diffuse Microglia Activation
Very low mutation burden is a feature of inflamed recurrent glioblastomas responsive to cancer immunotherapy.
Viral infection of cells within the tumor microenvironment mediates antitumor immunotherapy via selective TBK1-IRF3 signaling.
Enterovirus 2A<sup>pro</sup> Cleavage of the YTHDF m<sup>6</sup>A Readers Implicates YTHDF3 as a Mediator of Type I Interferon-Driven JAK/STAT Signaling.
CReP mediates selective translation initiation at the endoplasmic reticulum.
EXPERIMENTAL THERAPEUTICS AND PHARMACOLOGY