Area of research
Cellular and Molecular Neuroscience · Behavioral Neuroscience
Research interest
Research interests include Neuroscience and Neuropharmacology Research, Neurotransmitter Receptor Influence on Behavior, Stress Responses and Cortisol, and Epigenetics and DNA Methylation.
SIRT1 Coordinates Transcriptional Regulation of Neural Activity and Modulates Depression-Like Behaviors in the Nucleus Accumbens
Protocol for Xenium spatial transcriptomics studies using fixed frozen mouse brain sections
Early impairment of cortical circuit plasticity and connectivity in the 5XFAD Alzheimer’s disease mouse model
Disrupted Maturation of Prefrontal Layer 5 Neuronal Circuits in an Alzheimer’s Mouse Model of Amyloid Deposition
Shisa6 mediates cell-type specific regulation of depression in the nucleus accumbens
SIRT1 Mediates Depression-Like Behaviors in the Nucleus Accumbens
Role of Tet1 and 5-hydroxymethylcytosine in cocaine action
SIRT1-FOXO3a Regulate Cocaine Actions in the Nucleus Accumbens
Epigenetic basis of opiate suppression of Bdnf gene expression in the ventral tegmental area
Dishevelled-2 regulates cocaine-induced structural plasticity and Rac1 activity in the nucleus accumbens
Erratum to: Chronic cocaine-regulated epigenomic changes in mouse nucleus accumbens
Prefrontal Cortical Circuit for Depression- and Anxiety-Related Behaviors Mediated by Cholecystokinin: Role of ΔFosB
β-catenin mediates stress resilience through Dicer1/microRNA regulation
Locus-specific epigenetic remodeling controls addiction- and depression-related behaviors
Chronic cocaine-regulated epigenomic changes in mouse nucleus accumbens
Nucleus Accumbens-Specific Interventions in RGS9-2 Activity Modulate Responses to Morphine
Morphine and cocaine increase serum‐ and glucocorticoid‐inducible kinase 1 activity in the ventral tegmental area
Locus-specific epigenetic reprogramming for the study of reward pathology
Stress and CRF gate neural activation of BDNF in the mesolimbic reward pathway
Essential Role of SIRT1 Signaling in the Nucleus Accumbens in Cocaine and Morphine Action
Rapid regulation of depression-related behaviours by control of midbrain dopamine neurons
Rac1 is essential in cocaine-induced structural plasticity of nucleus accumbens neurons
BDNF Is a Negative Modulator of Morphine Action
Serum Response Factor and cAMP Response Element Binding Protein Are Both Required for Cocaine Induction of ΔFosB