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Eric Olinger

Newcastle University · GB
🔎 Find collaborators in Genetics · Molecular Biology →
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Area of research
Genetics · Molecular Biology
Research interest
Research interests include Kidney disease, Medicine, PKD1, Autosomal dominant polycystic kidney disease, Polycystic kidney disease, and Tamm–Horsfall protein.
h-index
citations
638
works
11
NIH funding
primary concept
email

Recent publications

Characterization of the Cystic Phenotype Associated with Monoallelic ALG8 and ALG9 Pathogenic Variants
Journal of the American Society of Nephrology 2025cited by 12position: middledoi
Clinical Spectrum and Prognosis of Atypical Autosomal Dominant Polycystic Kidney Disease Caused by Monoallelic Pathogenic Variants of IFT140
American Journal of Kidney Diseases 2024cited by 8position: middledoi
Progressive Kidney Failure by Angiotensinogen Inactivation in the Germline
Hypertension 2024cited by 0position: middledoi
Certain heterozygous variants in the kinase domain of the serine/threonine kinase NEK8 can cause an autosomal dominant form of polycystic kidney disease
Kidney International 2023cited by 38position: middledoi
#3166 RECOGNITION OF RENAL TUBULAR DYSGENESIS IN ADOLESCENT CKD BY BIALLELIC AGT VARIANTS REQUIRED BROAD GENETIC ANALYSIS
Nephrology Dialysis Transplantation 2023cited by 0position: middledoi
Progressive liver, kidney, and heart degeneration in children and adults affected by TULP3 mutations
The American Journal of Human Genetics 2022cited by 55position: middledoi
An intermediate-effect size variant in <i>UMOD</i> confers risk for chronic kidney disease
Proceedings of the National Academy of Sciences 2022cited by 42position: firstdoi
Monoallelic IFT140 pathogenic variants are an important cause of the autosomal dominant polycystic kidney-spectrum phenotype
The American Journal of Human Genetics 2021cited by 163position: middledoi
Clinical and genetic spectra of autosomal dominant tubulointerstitial kidney disease due to mutations in UMOD and MUC1
Kidney International 2020cited by 143position: firstdoi
Common Elements in Rare Kidney Diseases: Conclusions from a Kidney Disease: Improving Global Outcomes (KDIGO) Controversies Conference
Kidney International 2017cited by 59position: middledoi
The serine protease hepsin mediates urinary secretion and polymerisation of Zona Pellucida domain protein uromodulin
eLife 2015cited by 118position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

John A. Sayer · University of Newcastle Australia3 papers (2023–2024)Karl X. Knaup · Friedrich-Alexander-Universität Erlangen-Nürnberg2 papers (2023–2024)Antje Wiesener · Friedrich-Alexander-Universität Erlangen-Nürnberg2 papers (2023–2024)Mario Schiffer · Medizinische Hochschule Hannover2 papers (2023–2024)Michael S. Wiesener · Friedrich-Alexander-Universität Erlangen-Nürnberg2 papers (2023–2024)Katharina A.E. Broeker · University of Regensburg2 papers (2023–2024)Francesca Pasutto · Friedrich-Alexander-Universität Erlangen-Nürnberg2 papers (2023–2024)Florian J. Wopperer · Friedrich-Alexander-Universität Erlangen-Nürnberg2 papers (2023–2024)Olivier Devuyst · University Hospital Cologne2 papers (2015–2024)Karl F. Hilgers · Ludwig-Maximilians-Universität München2 papers (2023–2024)Romain Perrier · University of Lausanne1 papers (2015–2015) · 1 papers (2015–2015)Marko Poglitsch · University of Cape Town1 papers (2024–2024) · 1 papers (2015–2015) · 1 papers (2024–2024)Anne‐Sophie Denommé‐Pichon · University of Tübingen1 papers (2024–2024)Shuo Li · Shanghai Medical College of Fudan University1 papers (2015–2015)André Reis · University of Regensburg1 papers (2024–2024)Robin Shoemaker · University of Kentucky1 papers (2024–2024) · 1 papers (2024–2024)
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