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Miklós Sahin‐Tóth

U.S. National Science Foundation · US
Area of research
Surgery · Oncology
Research interest
Research interests include Pancreatitis Pathology and Treatment, Pancreatic and Hepatic Oncology Research, Pancreatic function and diabetes, and Gastrointestinal disorders and treatments.
h-index
55
citations
10,012
works
292
NIH funding
primary concept
email

Recent publications

Minigenes for heterologous expression of human and mouse cationic trypsinogen.
2026cited by 0position: contributordoi
Mutations of Gly277 in <i>CPA1</i>-related chronic pancreatitis: clinical clues for misfolding aetiology.
2026cited by 0position: contributordoi
Chymotrypsin Deficiency Accelerates Precancerous Remodeling of the Pancreas in KC Mice.
2026cited by 0position: contributordoi
AlphaMissense versus laboratory-based pathogenicity prediction of 13 novel missense <i>CPA1</i> variants from pancreatitis cases.
2025cited by 5position: contributordoi
Chronic pancreatitis in T7C140S mice with misfolding cationic trypsinogen mutant.
2025cited by 1position: contributordoi
Misfolding PRSS1 variant p.Ala61Val in a case of suspected intrauterine pancreatitis.
2025cited by 1position: contributordoi
Misfolding <i>CPA1</i> mutation accelerates precancerous pancreas lesions in <i>KC</i> mice.
2025cited by 0position: contributordoi
Chymotrypsin B2 (CTRB2) Deletion Variant Induces Endoplasmic Reticulum Stress but does not Increase Risk for Chronic Pancreatitis.
2025cited by 0position: contributordoi
Heterozygous Spink1 Deficiency Promotes Trypsin-dependent Chronic Pancreatitis in Mice
Cellular and Molecular Gastroenterology and Hepatology 2024cited by 18position: lastdoi
Heterozygous Spink1 Deficiency Promotes Trypsin-dependent Chronic Pancreatitis in Mice.
2024cited by 11position: contributordoi
Functional predictors of pathogenicity of missense <i>CPA1</i> variants in chronic pancreatitis.
2024cited by 10position: contributordoi
Secretagogue-induced pancreatitis in mice devoid of chymotrypsin.
2024cited by 6position: contributordoi
Novel chymotrypsin C (CTRC) variants from real-world genetic testing of pediatric chronic pancreatitis cases.
2024cited by 6position: contributordoi
Intron-mediated enhancement of SPINK1 expression for pancreatitis therapy.
2024cited by 2position: contributordoi
Trypsin Activity in Secretagogue-induced Murine Pancreatitis Is Solely Elicited by Cathepsin B and Does Not Mediate Key Pathologic Responses
Gastroenterology 2023cited by 14position: lastdoi
Substrate specificity of human chymotrypsin-like protease (CTRL) characterized by phage display-selected small-protein inhibitors.
2023cited by 8position: contributordoi
Mouse model of PRSS1 p.R122H-related hereditary pancreatitis highlights context-dependent effect of autolysis-site mutation.
2023cited by 6position: contributordoi
Risk of chronic pancreatitis in carriers of the c.180C>T (p.Gly60=) CTRC variant: case-control studies and meta-analysis.
2023cited by 6position: contributordoi
Modelling chronic pancreatitis as a complex genetic disease in mice.
2023cited by 3position: contributordoi
Preclinical testing of dabigatran in trypsin-dependent pancreatitis
JCI Insight 2022cited by 18position: lastdoi
Arg236 in human chymotrypsin B2 (CTRB2) is a key determinant of high enzyme activity, trypsinogen degradation capacity, and protection against pancreatitis
Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics 2022cited by 15position: middledoi
Loss-of-function variant in chymotrypsin like elastase 3B (CELA3B) is associated with non-alcoholic chronic pancreatitis
Pancreatology 2022cited by 15position: contributordoi
Preclinical testing of dabigatran in trypsin-dependent pancreatitis.
2022cited by 13position: contributordoi
Rate of Autoactivation Determines Pancreatitis Phenotype in Trypsinogen Mutant Mice.
2022cited by 13position: contributordoi
Novel p.G250A Mutation Associated with Chronic Pancreatitis Highlights Misfolding-Prone Region in Carboxypeptidase A1 (CPA1)
International Journal of Molecular Sciences 2022cited by 12position: lastdoi
Novel p.G250A Mutation Associated with Chronic Pancreatitis Highlights Misfolding-Prone Region in Carboxypeptidase A1 (CPA1).
2022cited by 12position: contributordoi
Chronic progression of cerulein-induced acute pancreatitis in trypsinogen mutant mice.
2022cited by 9position: contributordoi
Variants in the pancreatic CUB and zona pellucida-like domains 1 (CUZD1) gene in early-onset chronic pancreatitis - A possible new susceptibility gene.
2022cited by 8position: contributordoi
Bicarbonate defective CFTR variants increase risk for chronic pancreatitis: A meta-analysis.
2022cited by 7position: contributordoi
Defective binding of SPINK1 variants is an uncommon mechanism for impaired trypsin inhibition in chronic pancreatitis
Journal of Biological Chemistry 2021cited by 25position: lastdoi

Grants

No grants ingested yet.

Frequent collaborators

· 37 papers (2019–2026)András Szabó · University of Debrecen9 papers (2012–2021)Eszter Hegyi · University of Pecs8 papers (2015–2022)Eszter Hegyi · Elsevier, Inc.7 papers (2019–2023)Balázs Csaba Németh · University of Glasgow6 papers (2013–2022) · 6 papers (2019–2025) · 6 papers (2020–2025)Alexandra Demcsák · U.S. National Science Foundation6 papers (2019–2024)Péter Hegyi · Semmelweis University6 papers (2015–2020)Zsanett Jancsó · University of California, Los Angeles6 papers (2016–2022)Andrea Geisz · Boston University5 papers (2018–2023)Markus M. Lerch · Ludwig-Maximilians-Universität München4 papers (2014–2020) · 4 papers (2015–2019)Heiko Witt · Else Kröner-Fresenius-Stiftung4 papers (2012–2018)Péter Hegyi · Ludwig-Maximilians-Universität München4 papers (2013–2020)Zoltán Rakonczay · University of California System4 papers (2014–2020)Andrea Párniczky · University of Szeged4 papers (2015–2020)Evette S. Radisky · Sylvester Comprehensive Cancer Center3 papers (2013–2015) · 3 papers (2015–2020)Sandor Vajda · Department of Biomedical Engineering3 papers (2022–2025)