Area of research
Cellular and Molecular Neuroscience · Pathology and Forensic Medicine
Research interest
Research interests include Nerve injury and regeneration, Spinal Cord Injury Research, Neurogenesis and neuroplasticity mechanisms, and Pain Mechanisms and Treatments.
Chondroitin sulfate proteoglycans prevent immune cell phenotypic conversion and inflammation resolution via TLR4 in rodent models of spinal cord injury
Combined treatment with enteric neural stem cells and chondroitinase ABC reduces spinal cord lesion pathology
Refining rodent models of spinal cord injury
Moving beyond the glial scar for spinal cord repair
ErbB receptor signaling directly controls oligodendrocyte progenitor cell transformation and spontaneous remyelination after spinal cord injury
Neuromodulation in the restoration of function after spinal cord injury
Immune-evasive gene switch enables regulated delivery of chondroitinase after spinal cord injury
Optimising complementary soft tissue synchrotron X-ray microtomography for reversibly-stained central nervous system samples
Combined Transcriptomics, Proteomics and Bioinformatics Identify Drug Targets in Spinal Cord Injury
The soft mechanical signature of glial scars in the central nervous system
Neuregulin-1 controls an endogenous repair mechanism after spinal cord injury
High-throughput proteomics reveal alarmins as amplifiers of tissue pathology and inflammation after spinal cord injury
The Expression of Inflammatory Mediators in Bladder Pain Syndrome
Large animal and primate models of spinal cord injury for the testing of novel therapies
Chondroitinase gene therapy improves upper limb function following cervical contusion injury
Restoring function after spinal cord injury: towards clinical translation of experimental strategies
Large-Scale Chondroitin Sulfate Proteoglycan Digestion with Chondroitinase Gene Therapy Leads to Reduced Pathology and Modulates Macrophage Phenotype following Spinal Cord Contusion Injury
Regulation of IL-10 by Chondroitinase ABC Promotes a Distinct Immune Response following Spinal Cord Injury
Review: Manipulating the extracellular matrix and its role in brain and spinal cord plasticity and repair
Chondroitinase <scp>ABC</scp> promotes compensatory sprouting of the intact corticospinal tract and recovery of forelimb function following unilateral pyramidotomy in adult mice