Peter E.A. Ash

Interaction of tau with HNRNPA2B1 and N6-methyladenosine RNA mediates the progression of tauopathy

TIA1 potentiates tau phase separation and promotes generation of toxic oligomeric tau

Actionable Cytopathogenic Host Responses of Human Alveolar Type 2 Cells to SARS-CoV-2

BraInMap Elucidates the Macromolecular Connectivity Landscape of Mammalian Brain

Protein Interaction Network Biology in Neuroscience

5′ UTR variants in the quantitative trait gene <i>Hnrnph1</i> support reduced 5′ UTR usage and hnRNP H protein as a molecular mechanism underlying reduced methamphetamine sensitivity

A Mutation in <i>Hnrnph1</i> That Decreases Methamphetamine-Induced Reinforcement, Reward, and Dopamine Release and Increases Synaptosomal hnRNP H and Mitochondrial Proteins

RNA binding proteins co-localize with small tau inclusions in tauopathy

TIA1 regulates the generation and response to toxic tau oligomers

Heavy Metal Neurotoxicants Induce ALS-Linked TDP-43 Pathology

Reducing the RNA binding protein TIA1 protects against tau-mediated neurodegeneration in vivo

Dioxins and related environmental contaminants increase TDP-43 levels

Interaction of tau with the RNA-Binding Protein TIA1 Regulates tau Pathophysiology and Toxicity

Mutations in LRRK2 potentiate age-related impairment of autophagic flux

Aggregation-prone c9FTD/ALS poly(GA) RAN-translated proteins cause neurotoxicity by inducing ER stress

Pathological stress granules in Alzheimer’s disease

Antisense transcripts of the expanded C9ORF72 hexanucleotide repeat form nuclear RNA foci and undergo repeat-associated non-ATG translation in c9FTD/ALS