Julie D. Atkin

ALS/FTD-associated mutation in cyclin F inhibits ER-Golgi trafficking, inducing ER stress, ERAD and Golgi fragmentation

TDP-43 is a ubiquitylation substrate of the SCFcyclin F complex

Impaired NHEJ repair in amyotrophic lateral sclerosis is associated with TDP-43 mutations

CYLD is a causative gene for frontotemporal dementia – amyotrophic lateral sclerosis

The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins

Pathogenic mutation in the ALS/FTD gene, CCNF, causes elevated Lys48-linked ubiquitylation and defective autophagy

Casein kinase II phosphorylation of cyclin F at serine 621 regulates the Lys48-ubiquitylation E3 ligase activity of the SCF ^{(cyclin F)} complex

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

CCNF mutations in amyotrophic lateral sclerosis and frontotemporal dementia

NFκB is a central regulator of protein quality control in response to protein aggregation stresses via autophagy modulation

Extracellular wildtype and mutant SOD1 induces ER–Golgi pathology characteristic of amyotrophic lateral sclerosis in neuronal cells

Ataxin-2 interacts with FUS and intermediate-length polyglutamine expansions enhance FUS-related pathology in amyotrophic lateral sclerosis

Mutant FUS induces endoplasmic reticulum stress in amyotrophic lateral sclerosis and interacts with protein disulfide-isomerase