Area of research
Cellular and Molecular Neuroscience · Cognitive Neuroscience
Research interest
Research interests include Neuroscience and Neuropharmacology Research, Neural dynamics and brain function, Neurological disorders and treatments, and Photoreceptor and optogenetics research.
Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration.
Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration
Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration
Author response: Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration
Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration
Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration
Author response: Chronic hyperactivation of midbrain dopamine neurons causes preferential dopamine neuron degeneration
Dopamine subsystems that track internal states
Frontostriatal Projections Regulate Innate Avoidance Behavior
Thermal constraints on in vivo optogenetic manipulations
Thermal constraints on in vivo optogenetic manipulations.
Locomotor suppression by a monosynaptic amygdala to brainstem circuit
A Genetically Encoded Fluorescent Sensor Enables Rapid and Specific Detection of Dopamine in Flies, Fish, and Mice
Fast-Spiking Interneurons Supply Feedforward Control of Bursting, Calcium, and Plasticity for Efficient Learning
A Subpopulation of Striatal Neurons Mediates Levodopa-Induced Dyskinesia
Cell-Type-Specific Control of Brainstem Locomotor Circuits by Basal Ganglia
Pathway-Specific Remodeling of Thalamostriatal Synapses in Parkinsonian Mice
Activation of Direct and Indirect Pathway Medium Spiny Neurons Drives Distinct Brain-wide Responses
Parkinsonism Driven by Antipsychotics Originates from Dopaminergic Control of Striatal Cholinergic Interneurons
Cortico–Basal Ganglia Circuit Function in Psychiatric Disease
Optogenetics: 10 years after ChR2 in neurons—views from the community
Reassessing Models of Basal Ganglia Function and Dysfunction
Striatal Cholinergic Interneurons Drive GABA Release from Dopamine Terminals
Striatal Cholinergic Neurotransmission Requires VGLUT3
Differential Innervation of Direct- and Indirect-Pathway Striatal Projection Neurons
Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β
Control of Basal Ganglia Output by Direct and Indirect Pathway Projection Neurons
Inhibitory Interneuron Deficit Links Altered Network Activity and Cognitive Dysfunction in Alzheimer Model
Distinct roles for direct and indirect pathway striatal neurons in reinforcement
Direct Reprogramming of Mouse and Human Fibroblasts into Multipotent Neural Stem Cells with a Single Factor