Area of research
Behavioral Neuroscience · Endocrinology, Diabetes and Metabolism
Research interest
Research interests include Stress Responses and Cortisol, Hormonal and reproductive studies, Tryptophan and brain disorders, and Neurotransmitter Receptor Influence on Behavior.
Esketamine alleviates depressive-like behavior in neuropathic pain mice through the METTL3-GluA1 pathway
Repetitive Transcranial Magnetic Stimulation Induces Cognitive Recovery in Alzheimer's Disease via <scp>GABAergic</scp> Neuron Activation of the Cx3cl1‐Cx3cr1 Axis
Edaravone Dexborneol ameliorates the cognitive deficits of APP/PS1 mice by inhibiting TLR4/MAPK signaling pathway via upregulating TREM2
Testosterone deficiency worsens mitochondrial dysfunction in APP/PS1 mice
Repetitive Transcranial Magnetic Stimulation-Mediated Neuroprotection in the 5xFAD Mouse Model of Alzheimer’s Disease Through GABRG2 and SNAP25 Modulation
Hydralazine inhibits neuroinflammation and oxidative stress in APP/PS1 mice via TLR4/NF-κB and Nrf2 pathways
Nrf2 Deficiency Attenuates Testosterone Efficiency in Ameliorating Mitochondrial Function of the Substantia Nigra in Aged Male Mice
Testosterone ameliorates age-related brain mitochondrial dysfunction
Pentoxifylline Enhances Antioxidative Capability and Promotes Mitochondrial Biogenesis in D‐Galactose‐Induced Aging Mice by Increasing Nrf2 and PGC‐1<i>α</i> through the cAMP‐CREB Pathway
Parecoxib alleviates the motor behavioral decline of aged rats by ameliorating mitochondrial dysfunction in the substantia nigra via COX-2/PGE2 pathway inhibition
Testosterone enhances mitochondrial complex V function in the substantia nigra of aged male rats
Pentoxifylline enhances antioxidative capability and promotes mitochondrial biogenesis for improving age-related behavioral deficits
Testosterone propionate activated the Nrf2-ARE pathway in ageing rats and ameliorated the age-related changes in liver
LncRNAs down-regulate Myh1, Casr, and Mis18a expression in the Substantia Nigra of aged male rats
Amelioratory Effects of Testosterone Propionate on Age-related Renal Fibrosis via Suppression of TGF-β1/Smad Signaling and Activation of Nrf2-ARE Signaling
Testosterone Upregulates the Expression of Mitochondrial ND1 and ND4 and Alleviates the Oxidative Damage to the Nigrostriatal Dopaminergic System in Orchiectomized Rats
Testosterone Propionate Exacerbates the Deficits of Nigrostriatal Dopaminergic System and Downregulates Nrf2 Expression in Reserpine-Treated Aged Male Rats
Alleviation of Oxidative Damage and Involvement of Nrf2‐ARE Pathway in Mesodopaminergic System and Hippocampus of Status Epilepticus Rats Pretreated by Intranasal Pentoxifylline
The aberrantly expressed long non‐coding <scp>RNA</scp> in the substantia nigra and corpus striatum of Nrf2‐knockout mice
Enhancement of dopaminergic activity and region-specific activation of Nrf2-ARE pathway by intranasal supplements of testosterone propionate in aged male rats
Deficits in coordinated motor behavior and in nigrostriatal dopaminergic system ameliorated and VMAT2 expression up-regulated in aged male rats by administration of testosterone propionate
The effects of gonadectomy and binge-like ethanol exposure during adolescence on open field behaviour in adult male rats
Chronic testosterone propionate supplement could activated the Nrf2-ARE pathway in the brain and ameliorated the behaviors of aged rats