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Katalin Sándor

Karolinska Institutet · SE
Area of research
Physiology · Cellular and Molecular Neuroscience
Research interest
Research interests include Pain Mechanisms and Treatments, Neuropeptides and Animal Physiology, Ion Channels and Receptors, and Rheumatoid Arthritis Research and Therapies.
h-index
34
citations
3,137
works
86
NIH funding
primary concept
email

Recent publications

A subset of antibodies targeting citrullinated proteins confers protection from rheumatoid arthritis
Nature Communications 2023cited by 61position: middledoi
CD163+ macrophages monitor enhanced permeability at the blood–dorsal root ganglion barrier
The Journal of Experimental Medicine 2023cited by 50position: middledoi
Anti-satellite glia cell IgG antibodies in fibromyalgia patients are related to symptom severity and to metabolite concentrations in thalamus and rostral anterior cingulate cortex
Brain Behavior and Immunity 2023cited by 45position: middledoi
Insights into FcγR involvement in pain-like behavior induced by an RA-derived anti-modified protein autoantibody
Brain Behavior and Immunity 2023cited by 15position: middledoi
Passive transfer of fibromyalgia symptoms from patients to mice
Journal of Clinical Investigation 2021cited by 255position: middledoi
Disrupted function of lactate transporter <scp>MCT1</scp>, but not <scp>MCT4</scp>, in Schwann cells affects the maintenance of motor end‐plate innervation
Glia 2020cited by 39position: middledoi
Cartilage-binding antibodies induce pain through immune complex–mediated activation of neurons
The Journal of Experimental Medicine 2019cited by 102position: middledoi
Oxidative hotspots on actin promote skeletal muscle weakness in rheumatoid arthritis
JCI Insight 2019cited by 60position: middledoi
Exploring the transcriptome of resident spinal microglia after collagen antibody–induced arthritis
Pain 2018cited by 60position: middledoi
Cardiomyopathy, oxidative stress and impaired contractility in a rheumatoid arthritis mouse model
Heart 2018cited by 43position: middledoi
Autoantibodies to citrullinated proteins induce joint pain independent of inflammation via a chemokine-dependent mechanism
Annals of the Rheumatic Diseases 2015cited by 252position: middledoi
Collagen antibody–induced arthritis evokes persistent pain with spinal glial involvement and transient prostaglandin dependency
Arthritis & Rheumatism 2012cited by 113position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Camilla I. Svensson · Karolinska Institutet10 papers (2012–2023)Alexandra Jurczak · Karolinska Institutet6 papers (2018–2023)Nilesh M. Agalave · The University of Texas at Dallas4 papers (2012–2023)Emerson Krock · McGill University Health Centre3 papers (2021–2023)Azar Baharpoor · Karolinska Institutet3 papers (2012–2023)Jie Su · Karolinska Institutet3 papers (2012–2018)Matthew A. Hunt · Karolinska Institutet3 papers (2023–2023)Alex Bersellini Farinotti · Karolinska Institutet3 papers (2015–2023)Lars Klareskog · Karolinska University Hospital2 papers (2015–2023)Duygu B. Bas · Karolinska Institutet2 papers (2012–2015)Jon Lampa · Karolinska University Hospital2 papers (2018–2023)Angelica Sandström · Harvard University2 papers (2021–2023)Eva Kosek · Uppsala University2 papers (2021–2023)Jeanette Tour · Karolinska Institutet2 papers (2021–2023)Juan Miguel Jiménez‐Andrade · University of Arizona2 papers (2015–2023)Rikard Holmdahl · Uppsala University2 papers (2012–2015)Teresa Fernández-Zafra · Karolinska Institutet2 papers (2018–2018)Lisbet Haglund · McGill University Health Centre2 papers (2021–2023)Joana Menezes · Karolinska Institutet2 papers (2021–2023)Kutty Selva Nandakumar · Southern Medical University2 papers (2012–2015)