Area of research
Cell Biology · Developmental Neuroscience
Research interest
Research interests include Biology, Neuroscience, Cell biology, Microtubule, Kinesin, and Hereditary spastic paraplegia.
Veteran-derived cerebral organoids display multifaceted pathological defects in studies on Gulf War Illness
A cellular approach to understanding and treating Gulf War Illness
Boston biorepository, recruitment and integrative network (BBRAIN): A resource for the Gulf War Illness scientific community
Therapeutic Strategies for Mutant SPAST-Based Hereditary Spastic Paraplegia
Modeling gain-of-function and loss-of-function components of<i>SPAST</i>-based hereditary spastic paraplegia using transgenic mice
Tau Does Not Stabilize Axonal Microtubules but Rather Enables Them to Have Long Labile Domains
Hereditary spastic paraplegia: gain-of-function mechanisms revealed by new transgenic mouse
Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR)/Cas9‐mediated <i>kif15</i> mutations accelerate axonal outgrowth during neuronal development and regeneration in zebrafish
Mutant spastin proteins promote deficits in axonal transport through an isoform-specific mechanism involving casein kinase 2 activation
Pharmacologically increasing microtubule acetylation corrects stress‐exacerbated effects of organophosphates on neurons
Depletion of kinesin-12, a myosin-IIB-interacting protein, promotes migration of cortical astrocytes
Sliding of centrosome-unattached microtubules defines key features of neuronal phenotype
Vertebrate Fidgetin Restrains Axonal Growth by Severing Labile Domains of Microtubules
<scp>TPX</scp>2 regulates neuronal morphology through kinesin‐5 interaction
Pathogenic Mutation of Spastin Has Gain-of-Function Effects on Microtubule Dynamics
Kinesin‐12 influences axonal growth during zebrafish neural development
Polarity in Migrating Neurons Is Related to a Mechanism Analogous to Cytokinesis
Mitotic Motors Coregulate Microtubule Patterns in Axons and Dendrites