Area of research
Immunology · Molecular Biology
Research interest
Research interests include Receptor Mechanisms and Signaling, Rheumatoid Arthritis Research and Therapies, T-cell and B-cell Immunology, and Monoclonal and Polyclonal Antibodies Research.
Slope Extrusion Microchannel Integrating Terahertz Meta-Sensor Embedded in Cerebrospinal Fluid for Precise Diagnosis of PCNSIs
G protein-coupled receptor kinase 2 as a novel therapeutic target for gland fibrosis of Sjögren’s syndrome
GRK2 mediated degradation of SAV1 initiates hyperplasia of fibroblast-like synoviocytes in rheumatoid arthritis
Is Etiology a Key Factor for Regenerative Endodontic Treatment Outcomes?
EWSR1-induced circNEIL3 promotes glioma progression and exosome-mediated macrophage immunosuppressive polarization via stabilizing IGF2BP3
Gα12 and Gα13: Versatility in Physiology and Pathology
G protein-coupled receptor kinase type 2 and β-arrestin2: Key players in immune cell functions and inflammation
Attenuation of experimental osteoarthritis with human adipose-derived mesenchymal stem cell therapy: inhibition of the pyroptosis in chondrocytes
Triggers of Cardiovascular Diseases in Rheumatoid Arthritis
hIgDFc-Ig inhibits B cell function by regulating the BCR-Syk-Btk-NF-κB signalling pathway in mice with collagen-induced arthritis
Carvedilol induces biased β1 adrenergic receptor-nitric oxide synthase 3-cyclic guanylyl monophosphate signalling to promote cardiac contractility
GRK5 Controls SAP97-Dependent Cardiotoxic β <sub>1</sub> Adrenergic Receptor-CaMKII Signaling in Heart Failure
IgD-Fc-Ig fusion protein, a new biological agent, inhibits T cell function in CIA rats by inhibiting IgD-IgDR-Lck-NF-κB signaling pathways
BAFF promotes T cell activation through the BAFF-BAFF-R-PI3K-Akt signaling pathway
Molecular Mechanisms of T Cells Activation by Dendritic Cells in Autoimmune Diseases
Bidirectional Role of β2-Adrenergic Receptor in Autoimmune Diseases
Paroxetine alleviates T lymphocyte activation and infiltration to joints of collagen-induced arthritis
Insulin and β Adrenergic Receptor Signaling: Crosstalk in Heart
CP-25, a Novel Anti-inflammatory and Immunomodulatory Drug, Inhibits the Functions of Activated Human B Cells through Regulating BAFF and TNF-alpha Signaling and Comparative Efficacy with Biological Agents
Down-regulation of β-arrestin2 promotes tumour invasion and indicates poor prognosis of hepatocellular carcinoma
High‐fat diet induces protein kinase A and G‐protein receptor kinase phosphorylation of β<sub>2</sub>‐adrenergic receptor and impairs cardiac adrenergic reserve in animal hearts
Epigenetic Regulation of Phosphodiesterases 2A and 3A Underlies Compromised β-Adrenergic Signaling in an iPSC Model of Dilated Cardiomyopathy
Prednisone treatment inhibits the differentiation of B lymphocytes into plasma cells in MRL/MpSlac-lpr mice
Ginsenoside Metabolite Compound K Suppresses T-Cell Priming via Modulation of Dendritic Cell Trafficking and Costimulatory Signals, Resulting in Alleviation of Collagen-Induced Arthritis
Ginsenoside Metabolite Compound K Alleviates Adjuvant-Induced Arthritis by Suppressing T Cell Activation
Total glucosides of paeony inhibit the inflammatory responses of mice with allergic contact dermatitis by restoring the balanced secretion of pro-/anti-inflammatory cytokines
Ginsenoside metabolite compound K attenuates inflammatory responses of adjuvant-induced arthritis rats
Paeoniflorin inhibits inflammatory responses in mice with allergic contact dermatitis by regulating the balance between inflammatory and anti-inflammatory cytokines
BAFF/BAFF-R involved in antibodies production of rats with collagen-induced arthritis via PI3K-Akt-mTOR signaling and the regulation of paeoniflorin
Pro-apoptotic effect of Epigallo-catechin-3-gallate on B lymphocytes through regulating BAFF/PI3K/Akt/mTOR signaling in rats with collagen-induced arthritis