Area of research
Immunology · Physiology
Research interest
Research interests include IL-33, ST2, and ILC Pathways, Asthma and respiratory diseases, Immune Cell Function and Interaction, and Eosinophilic Esophagitis.
Prenatal inflammation remodels lung immunity and function by programming ILC2 hyperactivation
Progesterone amplifies allergic inflammation and airway pathology in association with higher lung ILC2 responses
Aryl Hydrocarbon Receptor Activation in Pulmonary Alveolar Epithelial Cells Limits Inflammation and Preserves Lung Epithelial Cell Integrity
Steady-state estradiol triggers a unique innate immune response to allergen resulting in increased airway resistance
IL-33 induces NF-κB activation in ILC2 that can be suppressed by in vivo and ex vivo 17β-estradiol
Neutralization of IL-33 modifies the type 2 and type 3 inflammatory signature of viral induced asthma exacerbation
SARS‐CoV‐2 innate effector associations and viral load in early nasopharyngeal infection