Area of research
Epidemiology · Hepatology
Research interest
Research interests include Liver Disease Diagnosis and Treatment, Hepatitis C virus research, Liver Diseases and Immunity, and Diet, Metabolism, and Disease.
Normalization of Oxygen Levels Induces a Metabolic Reprogramming in Livers Exposed to Intermittent Hypoxia Mimicking Obstructive Sleep Apnea.
Beneficial effects of hepatic cyclooxygenase-2 expression against cholestatic injury after common bile duct ligation in mice.
Assessment of Skeletal Muscle Alterations and Circulating Myokines in MASLD Patients: A Prospective Cohort Study
Increased let-7d-5p in non-alcoholic fatty liver promotes insulin resistance and is a potential blood biomarker for diagnosis.
Impaired Function of Solute Carrier Family 19 Leads to Low Folate Levels and Lipid Droplet Accumulation in Hepatocytes.
Mitochondrial respiratory chain activity is associated with severity, corticosteroid response and prognosis of alcoholic hepatitis.
Editorial: mitochondrial respiratory chain activity-a potential link with disease severity and treatment response in alcoholic hepatitis. Authors' reply.
Inhibition of ATG3 ameliorates liver steatosis by increasing mitochondrial function
Rhamnan sulfate reduces atherosclerotic plaque formation and vascular inflammation
Concerted regulation of non-alcoholic fatty liver disease progression by microRNAs in apolipoprotein E-deficient mice.
Daptomycin Plus Fosfomycin Versus Daptomycin Alone for Methicillin-resistant Staphylococcus aureus Severe Infection: Post Hoc Analysis Is Warranted.
Abstract 11489: Oral Rhamnan Sulfate Reduces Vascular Inflammation and Atherosclerotic Plaque Formation
Hypoxia-inducible factor 2α drives hepatosteatosis through the fatty acid translocase CD36.
A polymorphism in the Irisin-encoding gene (FNDC5) associates with hepatic steatosis by differential miRNA binding to the 3′UTR
Impaired autophagic flux is associated with increased endoplasmic reticulum stress during the development of NAFLD
Increased hepatic CD36 expression with age is associated with enhanced susceptibility to nonalcoholic fatty liver disease