Area of research
Immunology · Pathology and Forensic Medicine
Research interest
Research interests include T-cell and B-cell Immunology, Immunotherapy and Immune Responses, Immune Cell Function and Interaction, and Multiple Sclerosis Research Studies.
1523 Differential PERK Signaling Dysregulation in Alzheimer’s Disease Neurons and Glia
CAR-T Cell–Mediated B-Cell Depletion in Central Nervous System Autoimmunity
Targeting PGLYRP1 promotes antitumor immunity while inhibiting autoimmune neuroinflammation
Tim-3 adapter protein Bat3 acts as an endogenous regulator of tolerogenic dendritic cell function
Metabolic modeling of single Th17 cells reveals regulators of autoimmunity
In Silico Modeling of Metabolic State in Single Th17 Cells Reveals Novel Regulators of Inflammation and Autoimmunity
IL-11 Induces Encephalitogenic Th17 Cells in Multiple Sclerosis and Experimental Autoimmune Encephalomyelitis
DGAT1 inhibits retinol-dependent regulatory T cell formation and mediates autoimmune encephalomyelitis
The central vein sign and its clinical evaluation for the diagnosis of multiple sclerosis: a consensus statement from the North American Imaging in Multiple Sclerosis Cooperative
Dimethyl fumarate treatment induces adaptive and innate immune modulation independent of Nrf2
Tolerance checkpoint bypass permits emergence of pathogenic T cells to neuromyelitis optica autoantigen aquaporin-4
CD5L/AIM Regulates Lipid Biosynthesis and Restrains Th17 Cell Pathogenicity
Deletion of CTLA-4 on regulatory T cells during adulthood leads to resistance to autoimmunity
MHC class II–dependent B cell APC function is required for induction of CNS autoimmunity independent of myelin-specific antibodies
Defective sphingosine 1-phosphate receptor 1 (S1P1) phosphorylation exacerbates TH17-mediated autoimmune neuroinflammation
Fms-like tyrosine kinase 3 ligand-Fc chimera suppresses autoimmune demyelinating disease (P5228)
Induction and molecular signature of pathogenic TH17 cells
Bat3 promotes T cell responses and autoimmunity by repressing Tim-3–mediated cell death and exhaustion
Janus-like opposing roles of CD47 in autoimmune brain inflammation in humans and mice
Laquinimod, a Quinoline-3-Carboxamide, Induces Type II Myeloid Cells That Modulate Central Nervous System Autoimmunity
A Transgenic Model of Central Nervous System Autoimmunity Mediated by CD4+ and CD8+ T and B Cells