Area of research
Immunology and Allergy · Immunology
Research interest
Research interests include Cell Adhesion Molecules Research, Barrier Structure and Function Studies, Immune Response and Inflammation, and Neutrophil, Myeloperoxidase and Oxidative Mechanisms.
Claudin-23 reshapes epithelial tight junction architecture to regulate barrier function
Astrocytic junctional adhesion molecule-A regulates T-cell entry past the glia limitans to promote central nervous system autoimmune attack
Blocking integrin α4β7-mediated CD4 T cell recruitment to the intestine and liver protects mice from western diet-induced non-alcoholic steatohepatitis
Western diet‐induced increase in colonic bile acids compromises epithelial barrier in nonalcoholic steatohepatitis
Formyl peptide receptor 2 regulates monocyte recruitment to promote intestinal mucosal wound repair
Macrophage-dependent neutrophil recruitment is impaired under conditions of increased intestinal permeability in JAM-A-deficient mice
Macrophage-derived IL-10 mediates mucosal repair by epithelial WISP-1 signaling
Bioengineering Bacterially Derived Immunomodulants: A Therapeutic Approach to Inflammatory Bowel Disease
Loss of Junctional Adhesion Molecule A Promotes Severe Steatohepatitis in Mice on a Diet High in Saturated Fat, Fructose, and Cholesterol
IL-17A-mediated neutrophil recruitment limits expansion of segmented filamentous bacteria
Neutrophil interactions with epithelial-expressed ICAM-1 enhances intestinal mucosal wound healing
Norovirus in symptomatic and asymptomatic individuals: cytokines and viral shedding
Deposition of microparticles by neutrophils onto inflamed epithelium: a new mechanism to disrupt epithelial intercellular adhesions and promote transepithelial migration
Annexin A1–containing extracellular vesicles and polymeric nanoparticles promote epithelial wound repair
Cutting Edge: IL-36 Receptor Promotes Resolution of Intestinal Damage
Inflammation-induced desmoglein-2 ectodomain shedding compromises the mucosal barrier
Human norovirus infection and the acute serum cytokine response
HNF4α Regulates Claudin-7 Protein Expression during Intestinal Epithelial Differentiation
Targeting of Neutrophil Lewis X Blocks Transepithelial Migration and Increases Phagocytosis and Degranulation
Proinflammatory cytokine-induced tight junction remodeling through dynamic self-assembly of claudins
JAM-related proteins in mucosal homeostasis and inflammation
Interleukin‐4 and interleukin‐13 compromise the sinonasal epithelial barrier and perturb intercellular junction protein expression
Galectin-3 Regulates Desmoglein-2 and Intestinal Epithelial Intercellular Adhesion
Junctional Adhesion Molecule A Promotes Epithelial Tight Junction Assembly to Augment Lung Barrier Function
Epithelial adhesion molecules and the regulation of intestinal homeostasis during neutrophil transepithelial migration
<i>Trans-</i>dimerization of JAM-A regulates Rap2 and is mediated by a domain that is distinct from the<i>cis-</i>dimerization interface
IFNγ-induced suppression of β-catenin signaling: evidence for roles of Akt and 14.3.3ζ
The Role of Polymorphonuclear Leukocyte Trafficking in the Perpetuation of Inflammation During Inflammatory Bowel Disease
JAM-A associates with ZO-2, afadin, and PDZ-GEF1 to activate Rap2c and regulate epithelial barrier function
Transmigrated neutrophils in the intestinal lumen engage ICAM-1 to regulate the epithelial barrier and neutrophil recruitment