Area of research
Molecular Biology · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Mitochondrial Function and Pathology, Cardiovascular Function and Risk Factors, Adipose Tissue and Metabolism, and Ubiquitin and proteasome pathways.
Guardians of the Mitochondria: Space Mitochondria 2.0 Systemic Analysis Reveals Bioenergetic Dysregulation Across Species
Cosmic kidney disease: an integrated pan-omic, physiological and morphological study into spaceflight-induced renal dysfunction
Viral afterlife: SARS-CoV-2 as a reservoir of immunomimetic peptides that reassemble into proinflammatory supramolecular complexes
Aging and putative frailty biomarkers are altered by spaceflight
Lethal COVID-19 associates with RAAS-induced inflammation for multiple organ damage including mediastinal lymph nodes
Core mitochondrial genes are down-regulated during SARS-CoV-2 infection of rodent and human hosts
A comprehensive SARS-CoV-2 and COVID-19 review, Part 2: host extracellular to systemic effects of SARS-CoV-2 infection
Scientific Business Abstracts
Ubiquitin ligase STUB1 destabilizes IFNγ-receptor complex to suppress tumor IFNγ signaling
System-wide transcriptome damage and tissue identity loss in COVID-19 patients
The interplay between lncRNAs, RNA-binding proteins and viral genome during SARS-CoV-2 infection reveals strong connections with regulatory events involved in RNA metabolism and immune response
Role of miR-2392 in driving SARS-CoV-2 infection
Comprehensive Multi-omics Analysis Reveals Mitochondrial Stress as a Central Biological Hub for Spaceflight Impact
Content and Performance of the MiniMUGA Genotyping Array: A New Tool To Improve Rigor and Reproducibility in Mouse Research
Circulating miRNA Spaceflight Signature Reveals Targets for Countermeasure Development
Increasing Cardiomyocyte Atrogin-1 Reduces Aging-Associated Fibrosis and Regulates Remodeling in Vivo
Using Genetic Technologies To Reduce, Rather Than Widen, Health Disparities
The ubiquitin ligase MuRF1 regulates PPARα activity in the heart by enhancing nuclear export via monoubiquitination
Ataxia and hypogonadism caused by the loss of ubiquitin ligase activity of the U box protein CHIP