Area of research
Food Science · Cellular and Molecular Neuroscience
Research interest
Research focused on Metabolomics and Acrylamide, with related work in Inflammasome, Reactive oxygen species, Apoptosis. Notable publications include 'Metabolomics analysis of serum from subjects after occupational exposure to acrylamide using UPLC-MS', 'Nickel-refining fumes induce NLRP3 activation dependent on mitochondrial damage and ROS production in Beas-2B cells', and 'A urinary metabolomic study from subjects after long-term occupational exposure to low concentration acrylamide using UPLC-QTOF/MS'.
Fatty acid metabolism-derived prognostic model for lung adenocarcinoma: unraveling the link to survival and immune response
Hybrid construction of tissue-engineered nerve graft using skin derived precursors induced neurons and Schwann cells to enhance peripheral neuroregeneration
Reduction of eEF2 kinase alleviates the learning and memory impairment caused by acrylamide
Protective effect of resveratrol on nickel-refining fumes-induced inflammatory damage
Stimulating the expression of sphingosine kinase 1 (SphK1) is beneficial to reduce acrylamide-induced nerve cell damage
A urinary metabolomic study from subjects after long-term occupational exposure to low concentration acrylamide using UPLC-QTOF/MS
Nickel-refining fumes induce NLRP3 activation dependent on mitochondrial damage and ROS production in Beas-2B cells
Nickel-smelting fumes induce mitochondrial damage and apoptosis, accompanied by decreases in viability, in NIH/3T3 cells
Metabolomics analysis of serum from subjects after occupational exposure to acrylamide using UPLC-MS
[Nickel exposure to A549 cell damage and L-ascorbic acid interference effect].
PubMed 2015cited by 3position: middle