Area of research
Molecular Biology · Cell Biology
Research interest
Research interests include Proteoglycans and glycosaminoglycans research, Fibroblast Growth Factor Research, Estrogen and related hormone effects, and HER2/EGFR in Cancer Research.
Chemotherapy-Induced Sperm RNAs Disrupt Embryogenesis and Underlie Reproductive Outcomes in a Pre-Clinical Model of Childhood Cancer Survivors
Pancreatic cancer cell-intrinsic transglutaminase-2 promotes T cell suppression through microtubule-dependent secretion of immunosuppressive cytokines.
Circulating cell-free DNA methylation patterns indicate cellular sources of allograft injury after liver transplant.
Targeting TEAD in cancer.
Differential Effects of IL4I1 Protein on Lymphocytes From Healthy and Multiple Sclerosis Patients.
Senescence Inducers and Senolytics – Pathways and Drugs Targeting Cellular Senescence
TEAD inhibition alters the lung immune microenvironment and attenuates metastasis
A pancreatic cancer organoid-in-matrix platform shows distinct sensitivities to T cell killing.
The Role of Aging and Senescence in Immune Checkpoint Inhibitor Response and Toxicity.
Tumor specimen cold ischemia time impacts molecular cancer drug target discovery.
The Role of Aging and Senescence in Immune Checkpoint Inhibitor Response and Toxicity
The Role of Aging and Senescence in Immune Checkpoint Inhibitor Response and Toxicity
Circulating, cell-free methylated DNA indicates cellular sources of allograft injury after liver transplant
Circulating cell-free methylated DNA reveals tissue-specific, cellular damage from radiation treatment
Pleiotrophin drives a prometastatic immune niche in breast cancer.
Stromal Senescence following Treatment with the CDK4/6 Inhibitor Palbociclib Alters the Lung Metastatic Niche and Increases Metastasis of Drug-Resistant Mammary Cancer Cells.
AIB1/SRC-3/NCOA3 function in estrogen receptor alpha positive breast cancer.
Circulating cell-free methylated DNA reveals tissue-specific, cellular damage from radiation treatment.
Loss of ANCO1 Expression Regulates Chromatin Accessibility and Drives Progression of Early-Stage Triple-Negative Breast Cancer.
Subpopulations of AIB1 isoform-expressing breast cancer cells enable invasion and metastasis.
Figure S1 from An AIB1 Isoform Alters Enhancer Access and Enables Progression of Early-Stage Triple-Negative Breast Cancer
Supplemental Figure 6 from Short- and Long-Term Effects of CDK4/6 Inhibition on Early-Stage Breast Cancer
Supplementary Methods from Short- and Long-Term Effects of CDK4/6 Inhibition on Early-Stage Breast Cancer
Supplemental Figure 3 from Short- and Long-Term Effects of CDK4/6 Inhibition on Early-Stage Breast Cancer
Supplemental Figure 6 from Short- and Long-Term Effects of CDK4/6 Inhibition on Early-Stage Breast Cancer
Table S1 from An AIB1 Isoform Alters Enhancer Access and Enables Progression of Early-Stage Triple-Negative Breast Cancer
Figure S5 from An AIB1 Isoform Alters Enhancer Access and Enables Progression of Early-Stage Triple-Negative Breast Cancer
Table S2 from An AIB1 Isoform Alters Enhancer Access and Enables Progression of Early-Stage Triple-Negative Breast Cancer
Data from Short- and Long-Term Effects of CDK4/6 Inhibition on Early-Stage Breast Cancer
Supplemental Figure 7 from Short- and Long-Term Effects of CDK4/6 Inhibition on Early-Stage Breast Cancer