Area of research
Immunology · Immunology and Allergy
Research interest
Research interests include Neutrophil, Myeloperoxidase and Oxidative Mechanisms, Cell Adhesion Molecules Research, Hidradenitis Suppurativa and Treatments, and Immune Response and Inflammation.
Dysregulation of U12-Type Splicing in Lupus Neutrophils.
Anti-Carbamylated Protein Antibodies Stabilize Carbamylated Histone H3 to Promote Synovial Activation and Neutrophil Extracellular Trap-Mediated Bone Loss in Rheumatoid Arthritis.
IgA autoantibodies promote inflammation, Th17 polarization and fibrotic responses in hidradenitis suppurativa.
Hidradenitis suppurativa: Intentional efforts to unravel precise mechanisms.
Role of STING Deficiency in Amelioration of Mouse Models of Lupus and Atherosclerosis.
Neutrophil extracellular traps activate Notch-γ-secretase signaling in hidradenitis suppurativa.
Soluble CD13 is a potential mediator of neutrophil-induced thrombogenic inflammation in SARS-CoV-2 infection
Standardized Protocols for Clinical and Histopathological Characterization of Hidradenitis Suppurativa Tissue Specimens
Mosquito salivary apyrase regulates blood meal hemostasis and facilitates malaria parasite transmission.
The Aconitate Decarboxylase 1/Itaconate Pathway Modulates Immune Dysregulation and Associates with Cardiovascular Disease Markers and Disease Activity in Systemic Lupus Erythematosus.
Neutrophil extracellular traps and extracellular histones potentiate IL-17 inflammation in periodontitis.
Neutrophil extracellular trap-associated carbamylation and histones trigger osteoclast formation in rheumatoid arthritis.
Low-density granulocytes in systemic autoimmunity and autoinflammation.
Apoptosis-induced nuclear expulsion in tumor cells drives S100a4-mediated metastatic outgrowth through the RAGE pathway.
Coexisting autoantibodies against transcription factor Sp4 are associated with decreased cancer risk in patients with dermatomyositis with anti-TIF1γ autoantibodies.
Neutralizing Anti‒DNase 1 and ‒DNase 1L3 Antibodies Impair Neutrophil Extracellular Traps Degradation in Hidradenitis Suppurativa
Constitutively active Lyn kinase causes a cutaneous small vessel vasculitis and liver fibrosis syndrome.
<i>Anopheles</i> salivary apyrase regulates blood meal hemostasis and drives malaria parasite transmission
Autoantibodies Present in Hidradenitis Suppurativa Correlate with Disease Severity and Promote the Release of Proinflammatory Cytokines in Macrophages
Modulation of the Itaconate Pathway Attenuates Murine Lupus.
Multicenter analysis of neutrophil extracellular trap dysregulation in adult and pediatric COVID-19.
Multicenter analysis of neutrophil extracellular trap dysregulation in adult and pediatric COVID-19
Autoantibodies Present in Hidradenitis Suppurativa Correlate with Disease Severity and Promote the Release of Proinflammatory Cytokines in Macrophages
Somatic Mutations in <i>UBA1</i> and Severe Adult-Onset Autoinflammatory Disease
Sex differences in neutrophil biology modulate response to type I interferons and immunometabolism.
Neutrophil extracellular traps mediate articular cartilage damage and enhance cartilage component immunogenicity in rheumatoid arthritis.
Neutrophil dysregulation is pathogenic in idiopathic inflammatory myopathies
Neutrophil dysregulation is pathogenic in idiopathic inflammatory myopathies.
Neutrophil-mediated carbamylation promotes articular damage in rheumatoid arthritis.
Technical comment on "Synovial fibroblast-neutrophil interactions promote pathogenic adaptive immunity in rheumatoid arthritis".