Area of research
Molecular Biology · Plant Science
Research interest
Research interests include Cell biology, Chemistry, Endocrinology, Internal medicine, Vimentin, and Parkinson's disease.
Construction of a Heterotrophic Nitrification–Aerobic Denitrification Composite Microbial Consortium and Its Bioaugmentation Role in Wastewater Treatment
Cordycepin promotes autophagic degradation of α-synuclein via CacyBP/SIP activation for ameliorating olfactory dysfunction against Parkinson's disease
Itchy E3 Ubiquitin Ligase-Mediated Ubiquitination of Ferritin Light Chain Contributes to Endothelial Ferroptosis in Atherosclerosis
Naringenin ameliorates vascular senescence and atherosclerosis involving SIRT1 activation
Olfactory dysfunction and its related molecular mechanisms in Parkinson’s disease
MDM2-Mediated Ubiquitination of RXRβ Contributes to Mitochondrial Damage and Related Inflammation in Atherosclerosis
2, 3, 4′, 5-tetrahydroxystilbene-2-0-β-d Glycoside Attenuates Age- and Diet-Associated Non-Alcoholic Steatohepatitis and Atherosclerosis in LDL Receptor Knockout Mice and Its Possible Mechanisms
On-Demand Versatile Prodrug Nanomicelle for Tumor-Specific Bioimaging and Photothermal-Chemo Synergistic Cancer Therapy
Salusin-α attenuates hepatic steatosis and atherosclerosis in high fat diet-fed low density lipoprotein receptor deficient mice
RETRACTED: NOSH-NBP, a Novel Nitric Oxide and Hydrogen Sulfide- Releasing Hybrid, Attenuates Ischemic Stroke-Induced Neuroinflammatory Injury by Modulating Microglia Polarization
Raf-1/CK2 and RhoA/ROCK signaling promote TNF-α-mediated endothelial apoptosis via regulating vimentin cytoskeleton
Current pharmacological developments in 2,3,4′,5-tetrahydroxystilbene 2-O-β-D-glucoside (TSG)
2,3,4′,5-tetrahydroxystilbene-2-<i>O</i>-β-<scp>d</scp>-glycoside attenuates atherosclerosis in apolipoprotein E-deficient mice: role of reverse cholesterol transport
The inhibition of macrophage foam cell formation by tetrahydroxystilbene glucoside is driven by suppressing vimentin cytoskeleton
Effect of Salusin-ß on Peroxisome Proliferator-Activated Receptor Gamma Gene Expression in Vascular Smooth Muscle Cells and its Possible Mechanism
Prostacyclin Analogue Beraprost Inhibits Cardiac Fibroblast Proliferation Depending on Prostacyclin Receptor Activation through a TGF β-Smad Signal Pathway
Salusin-β Induces Smooth Muscle Cell Proliferation by Regulating Cyclins D1 and E Expression Through MAPKs Signaling Pathways
Inhibitory effects of 2,3,4′,5-tetrahydroxystilbene-2-O-β-D-glucoside on angiotensin II-induced proliferation of vascular smooth muscle cells
The effect of 2,3,4′,5-tetrahydroxystilbene-2-0-β-d glucoside on neointima formation in a rat artery balloon injury model and its possible mechanisms